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INCIDENT CARDIOVASCULAR AND VENOUS THROMBOEMBOLIC EVENTS IN AUTOANTIBODY-DEFINED SLE CLUSTERS

2025· article· en· W4410513028 on OpenAlexvenueno aff
Sara Ferrigno, Elizabeth V. Arkema, Iva Gunnarsson, Agneta Zickert, Lina-Marcela Díaz-Gallo, Elisabet Svenungsson

Bibliographic record

VenueThe Journal of Rheumatology · 2025
Typearticle
Languageen
FieldMedicine
TopicSystemic Lupus Erythematosus Research
Canadian institutionsnot available
Fundersnot available
KeywordsMedicineAutoantibodyVenous thromboembolic diseaseVenous thromboembolismVenous thrombosisInternal medicineCardiologyThrombosisAntibodyImmunology

Abstract

fetched live from OpenAlex

PV197 / #398 Poster Topic: AS22 - SLE Heterogeneity Background/Purpose Patients with SLE are at high risk of cardiovascular (CV) and venous thromboembolic (VTE) events. Previously, we identified 4 autoantibody-defined SLE clusters,[1] associated with different HLA-DRB1 genotypes, clinical manifestations and cytokine patterns. We aimed to compare incidence rates of CV and VTE events between the clusters and to compare each cluster to a general population control group. Methods Unsupervised clustering, based on 13 autoantibodies, clustered SLE patients into 4 clusters: Cluster 1 was dominated (>50% positive) by anti-SSA/SSB, Cluster 2 by anti-nucleosome/Sm/RNP/dsDNA, Cluster 3 by aPL and Cluster 4 by negativity to the 13 autoantibodies. Information on vascular outcomes was collected through ICD codes from the National Patient Register. Controls from the Total Population Register were matched 10:1 on birth year, sex and residence to each patient. Subjects with a vascular event before enrollment were excluded. Incidence rates (IR) were calculated for 1000 person-years and 95% CI were calculated using the Poisson distribution. Age-adjusted Hazard Ratios (HR) and 95% CI from Cox proportional hazards models estimated relative risks of incident vascular events. Results 461 SLE patients were enrolled, mean follow-up of 12.24±5.6 years. SLE patients in cluster 2 were younger at inclusion and younger at first CV and VTE event. Cluster 3 had the highest crude IR for all the outcomes. The risk of major adverse CV events (MACE) in cluster 3 was almost twice as high compared to cluster 4 (Table 1). Moreover, cluster 3 had more than 2.5 higher risk of cerebrovascular events and VTE than cluster 1. Cluster 2 had similarly high HR for heart failure and VTE. Notably, HR for ischemic heart disease did not differ between cluster 4 and controls (Table 1). Table 1. Incidence rates and Hazard ratios (HR) for vascular outcomes between SLE clusters and between each cluster and general population controls. MACE: major adverse cardiovascular events; IHD:ischemic heart disease; VTE:venous thromboembolism; NP:not performed. *Age-adjusted Cox regression models. MACE= IHD+ischemic cerebrovascular events+peripheral arterial thrombosis/embolism+heart failure+death due to CV events; Cerebrovascular events=ischemic cerebrovascular events+cerebral hemorrhages; VTE=deep venous thrombosis+pulmonary embolism. Conclusions In SLE, incidence of MACE and VTE differs between autoantibody-defined clusters, with the highest incidence observed in the aPL positive and the lowest incidence in the autoantibody negative patients. References: [1.] Diaz-Gallo LM. ACR Open Rheum 2022;4(1):27-39.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.002
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.294
Teacher spread0.279 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2025
Admission routes1
Has abstractyes

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