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Record W4411625965 · doi:10.1101/2025.06.23.25330155

Genomic dimensions deconstruct the clinical heterogeneity of bipolar disorder

2025· preprint· en· W4411625965 on OpenAlexaff
Tracey van der Veen, Markos Tesfaye, Jessica Yang, Toni Boltz, Friederike S. David, Shane Crinion, Maria Koromina, Till F. M. Andlauer, Tim B. Bigdeli, Brandon J. Coombes, Tiffany A. Greenwood, Georgia Panagiotaropoulou, Nadine Parker, Heejong Sung, Nicholas Bass, Jonathan R. I. Coleman, José Guzmán‐Parra, János Kálmán, Caroline C. McGrouther, Brittany L. Mitchell, Aaditya V. Rangan, Katie Scott, Alexey Shadrin, Daniel J. Smith, Annabel Vreeker, Kristina Adorjan, Diego Albani, Silvia Alemany, Ney Alliey‐Rodriguez, Anastasia Antoniou, Michael Bauer, Eva C. Beins, Marco P. Boks, Rosa Bosch, Ben Brumpton, Nathalie Brunkhorst-Kanaan, Monika Budde, William Byerley, Judit Cabana‐Domínguez, Murray J. Cairns, Bernardo Carpiniello, Miguel Casas, Pablo Cervantes, Chris Chatzinakos, Toni‐Kim Clarke, Isabelle Claus, Cristiana Cruceanu, Alfredo B. Cuéllar‐Barboza, Piotr M. Czerski, Konstantinos Dafnas, Anders M. Dale, Nina Dalkner, J. Raymond DePaulo, Franziska Degenhardt, Srdjan Djurovic, Valentina Escott‐Price, Ayman H. Fanous, Frederike T. Fellendorf, I. Nicol Ferrier, Liz Forty, Josef Frank, Oleksandr Frei, Nelson B. Freimer, Julie Garnham, Ian R. Gizer, Scott D. Gordon, Katherine Gordon‐Smith, Tim Hahn, L Evatt Marian, Arvid Harder, Martin Hautzinger, Urs Heilbronner, Dennis Hellgren, Stefan Herms, Ian B. Hickie, Per Hoffmann, Peter Holmans, Stéphane Jamain, Lina Jönsson, James L. Kennedy, Sarah Kittel‐Schneider, James A. Knowles, Elise Koch, Manolis Kogevinas, Thorsten M. Kranz, Steven A. Kushner, Catharina Lavebratt, Jacob Lawrence, Markus Leber, Penelope A. Lind, Susanne Lucae, Martin Lundberg, Donald J. MacIntyre, Wolfgang Maier, Adam X. Maihofer, Dolores Malaspina, Mirko Manchia, Eirini Maratou, Lina Martinsson, Melvin G. McInnis, James McKay, Helena Medeiros, Andreas Meyer‐Lindenberg, Vincent Millischer, Derek W. Morris, Paraskevi Moutsatsou, Thomas W. Mühleisen, Claire O. ’Donovan, Catherine M. Olsen, Sergi Papiol, Antonio F. Pardiñas, Amy Perry, Andrea Pfennig, Claudia Pisanu, James B. Potash, Digby Quested, Mark Hyman Rapaport, Eline J. Regeer, John P. Rice, Margarita Rivera, Eva C. Schulte, Fanny Senner, Paul D. Shilling, Lisa Sindermann, Lea Sirignano, Dan Siskind, Claire Slaney, Olav B. Smeland, Janet L. Sobell, María Soler Artigas, Dan J. Stein, Frederike Stein, Beata Świątkowska, Jackson G. Thorp, Claudio Toma, Leonardo Tondo, Paul A. Tooney, Marquis P. Vawter, Helmut Vedder, James Walters, Stephanie H. Witt, Allan H. Young, Peter P. Zandi, Lea Zillich, Rolf Adolfsson, Lars Alfredsson, Lena Backlund, Bernhard T. Baune, Frank Bellivier, Susanne Bengesser, Wade H. Berrettini, Joanna M. Biernacka, Douglas Blackwood, Michael Boehnke, Gerome Breen, Vaughan J. Carr, Stanley V. Catts, Sven Cichon, Aiden Corvin, Nicholas Craddock, Udo Dannlowski, Dimitris Dikeos, Tõnu Esko, Bruno Étain, Panagiotis Ferentinos, Mark A. Frye, Janice M. Fullerton, Micha Gawlik, Elliot S. Gershon, Fernando S. Goes, Melissa J. Green, Joanna Hauser, Frans Henskens, Jens Hjerling‐Leffler, Ian Jones, Lisa Jones, René S. Kahn, John R. Kelsoe, Tilo Kircher, George Kirov, Mikael Landén, Marion Leboyer, Melanie Lenger, Qingqin S. Li, Jolanta Lissowska, Carmel Loughland, Jurjen J. Luykx, Nicholas G. Martin, Carol A. Mathews, Fermín Mayoral, Susan L. McElroy, Andrew M. McIntosh, Sarah E. Medland, Ingrid Melle, Philip B. Mitchell, Gunnar Morken, R Myers, Bertram Müller‐Myhsok, Benjamin M. Neale, Caroline M. Nievergelt, John I. Nürnberger, Markus M. Nöthen, Michael O‘Donovan, Ketil J. Øedegaard, Tomas Olsson, Michael J. Owen, Sara A. Paciga, Christos Pantelis, Carlos N. Pato, Michele T. Pato, George P. Patrinos, Joanna Pawlak, Roy H. Perlis, Josep Antoni Ramos‐Quiroga, Andreas Reif, Eva Z. Reininghaus, Marta Ribasés, Marcella Rietschel, Stephan Ripke, Guy A. Rouleau, Ulrich Schall, Martin Schalling, Peter R. Schofield, Thomas G. Schulze, Laura J. Scott, Alessandro Serretti, Jordan W. Smoller, Alessio Squassina, Eli A. Stahl, Eystein Stordal, Fabian Streit, Patrick F. Sullivan, Gustavo Turecki, Eduard Vieta, John B. Vincent, Irwin D. Waldman, Cynthia Shannon Weickert, Thomas W. Weickert, David C. Whiteman, Martin Alda, Roel A. Ophoff, Kevin S. O’Connell, Niamh Mullins, Andreas J. Forstner, Maria Grigoroiu‐Serbânescu, Howard J. Edenberg, Francis J. McMahon, Ole A. Andreassen, Arianna Di Florio, Andrew McQuillin

Bibliographic record

VenuemedRxiv · 2025
Typepreprint
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetic Associations and Epidemiology
Canadian institutionsMcGill UniversityDalhousie UniversityMontreal Neurological Institute and HospitalUniversity of TorontoCentre for Addiction and Mental HealthMcGill University Health Centre
FundersNational Institute of Mental HealthNational Institutes of Health
KeywordsNeuroscienceBipolar disorderImmune systemPsychologyBiologyImmunologyCognition

Abstract

fetched live from OpenAlex

Importance: The clinical heterogeneity of bipolar disorder (BD) is a major obstacle to improving diagnosis, predicting patient outcomes, and developing personalized treatments. A genetic approach is needed to deconstruct the disorder and uncover its fundamental biology. Previous genetic studies focusing on broad diagnostic categories have been limited in their ability to parse this complexity. Objective: To test the hypothesis that clinically distinct subphenotypes of BD are associated with different underlying common variant genetic architectures. Design Setting and Participants: This multicenter study included a primary genome-wide association study (GWAS) of up to 23,819 bipolar disorder (BD) cases and 163,839 controls. These results were integrated via multi-trait analysis of GWAS (MTAG) with external summary statistics for BD (59,287 cases; 781,022 controls) and schizophrenia (SCZ; 53,386 cases; 77,258 controls). Sample overlap was statistically accounted for. Main Outcomes and Measures: ), and functional, cell-type, and gene-expression pathway analyses. Results: ). Notably, the rapid-cycling subphenotype showed a unique signature of strong negative selection, a finding not observed in other subphenotypes. Conclusions and Relevance: The clinical heterogeneity of bipolar disorder appears to be defined by a complex and multi-layered genetic architecture. The presented findings provide an empirical framework that may advance psychiatric nosology beyond its current diagnostic boundaries. These results may also inform future research to identify targets for personalized interventions. The delineation of these genetically-informed dimensions offers specific, biologically-grounded hypotheses for subsequent therapeutic discovery. Establishing such a framework is an essential step toward refining diagnostic criteria and developing more effective, personalized treatments. This work lays the foundation for a transition from a uniform treatment model to the paradigm of precision psychiatry. Key Points: These findings provide a data-driven biological framework for bipolar disorder, guiding future research toward patient stratification and targeted therapeutics.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.003
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.003
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.001
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.030
GPT teacher head0.336
Teacher spread0.306 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2025
Admission routes1
Has abstractyes

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