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Record W4411960251 · doi:10.48048/tis.2025.9754

Apolipoprotein E (ApoE) Gene Polymorphism and Cardiovascular Risk in Type 2 Diabetic Patients: A Systematic Review

2025· review· en· W4411960251 on OpenAlexaboutno aff
A Thahira, D. Thirumal Kumar, Divya Roy, Swetapadma Pradhan, Yatindra Kumar, Arghadip Das, Ajit Sneha Shrikant Panvalkar, Sagar Harshad, V P Akshay, Delna N. Saraswathy

Bibliographic record

VenueTrends in Sciences · 2025
Typereview
Languageen
FieldMedicine
TopicDiabetes, Cardiovascular Risks, and Lipoproteins
Canadian institutionsnot available
Fundersnot available
KeywordsApolipoprotein EMedicinePolymorphism (computer science)Apolipoprotein BInternal medicineGeneticsGeneBioinformaticsBiologyAlleleDiseaseCholesterol

Abstract

fetched live from OpenAlex

Background: Apolipoprotein E gene (ApoE) polymorphism has been extensively studied in the context of lipid metabolism and cardiovascular disease (CVD) risk. Its association with Type 2 Diabetes Mellitus (T2DM) patients presents a unique subset of cardiovascular risk due to underlying metabolic changes. This systematic review aims to summarize and critically evaluate the available evidence on the impact of ApoE gene polymorphism on cardiovascular risk among T2DM patients. Methods: We used the PEO (Population, Exposure and Outcome) framework to develop our protocol, which is publicly available in the Open Science Framework (OSF) registries. Following this protocol, a systematic search was conducted in PubMed, SCOPUS, and Google Scholar to identify studies focusing on adult individuals (≥ 18 years old) diagnosed with Type 2 Diabetes Mellitus (T2DM) that reported on ApoE gene polymorphisms. Only studies published in English between 2014 and 2024 were included. Three authors independently assessed the quality of the eligible studies using the Newcastle-Ottawa Scale (NOS) for case-control, cohort, and cross-sectional designs and the qualified studies were selected for full-text screening and data extraction. Results: 16 studies satisfied the quality assessment using the Newcastle-Ottawa Scale (NOS) with satisfying NOS scores included in the final review, which comprised 1 cohort study, 2 cross-sectional studies, and 13 case-control studies. Across the studies, E3/E3 is the dominant genotype in both control and T2DM populations. E3/E4 and E4/E4 genotypes are found to be significantly elevated in T2DM cases with cardiovascular risk factors compared to controls. The Ɛ3 allele is the most common across all populations, in most studies. The Ɛ4 allele is consistently strongly associated with elevated levels of low-density lipoprotein (LDL-C), total cholesterol, and triglycerides. Conclusion: Apolipoprotein E gene polymorphism is an evident risk factor for cardiovascular complications and ApoE genotyping will be a valuable tool for stratifying T2DM patients based on cardiovascular risk. HIGHLIGHTS Apolipoprotein E gene polymorphisms exert significant influences on lipid metabolism and cardiovascular risk among type 2 diabetes patients (T2DM). The Ɛ4 allele contributes to increased levels of low-density lipoprotein (LDL-C), total cholesterol, and triglycerides in T2DM patients. Apolipoprotein E gene genotyping can aid in stratifying cardiovascular risk in T2DM patients. The E3/E4 and E4/E4 genotypes are more frequent in T2DM patients with cardiovascular complications. GRAPHICAL ABSTRACT

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.007
metaresearch head score (Gemma)0.028
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Systematic review · Consensus signal: Systematic review
GenreCandidate signal: Review · Consensus signal: Review
Teacher disagreement score0.008
Threshold uncertainty score0.035

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0070.028
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0080.006
Bibliometrics0.0080.010
Science and technology studies0.0010.001
Scholarly communication0.0020.002
Open science0.0020.001
Research integrity0.0020.001
Insufficient payload (model declined to judge)0.0040.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.031
GPT teacher head0.318
Teacher spread0.288 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designSystematic review
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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