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Abstract B042: Identifying single-cell transcriptomic signatures and changes underlying the evolution of fusion-negative rhabdomyosarcoma in Li-Fraumeni Syndrome mice

2025· article· en· W4412163760 on OpenAlexaffabout
Ashby Kissoondoyal, Paula R. Quaglietta, Brianne Laverty, David Malkin

Bibliographic record

VenueClinical Cancer Research · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer-related molecular mechanisms research
Canadian institutionsHospital for Sick Children
Fundersnot available
KeywordsLi–Fraumeni syndromeTranscriptomeRhabdomyosarcomaCancer researchBiologyFusion geneMedicineComputational biologyGeneticsPathologyGeneSarcomaMutationGene expression

Abstract

fetched live from OpenAlex

Abstract Soft tissue sarcomas (STS) comprise ∼7% of pediatric cancers, with over half classified as rhabdomyosarcoma (RMS), equating to approximately 4.5 cases per 1 million children. Germline mutations in the TP53 tumor suppressor gene (MutTP53), most notably in Li-Fraumeni Syndrome (LFS), represent the strongest heritable risk factor for RMS and are associated with poorer overall survival. Despite this, a transcriptomic signature specific to LFS-associated RMS (LFS-RMS) remains undefined. To address this, we performed droplet-based single-cell RNA sequencing (scRNA-seq; 10x Genomics) on RMS tumors and matched healthy muscle tissue from Trp53 R172H/WT mice, harboring a mutation analogous to the human TP53 R175H hotspot. Tumors were collected at endpoint from male (n = 2) and female (n = 3) mice. Cell types were identified through differential gene expression (DGE) analysis and annotated via consensus across machine learning-based tools. Trajectory analysis using Monocle3 revealed lineage transitions, while Generalized Additive Models (GAMs) identified genes associated with the progression from non-malignant to cancer-associated states. We observed consistent alterations in cell subtype proportions between tumor and healthy tissue, including a significant reduction in the M1/M2 macrophage proportion. Trajectory analysis identified key transcripts driving the transition from muscle-associated to cancer-associated fibroblasts and myogenic cells. Pathways enriched in GO analysis suggest that during the transition to cancer-associated phenotypes, cells in LFS-RMS adopt gene programs that reduce susceptibility to immune cell invasion in the tumor microenvironment. Our findings provide the first single-cell map of LFS-RMS, revealing dynamic transcriptional states and biomarkers of cells states associated with tumorigenesis. These insights pave the way for improved therapeutic approaches tailored to the complex cellular heterogeneity of LFS tumors. Further, these findings may broadly enhance RMS diagnostics and precision oncology approaches. Citation Format: Ashby Kissoondoyal, Paula R. Quaglietta, Brianne Laverty, Safa Majeed Grant, David Malkin. Identifying single-cell transcriptomic signatures and changes underlying the evolution of fusion-negative rhabdomyosarcoma in Li-Fraumeni Syndrome mice [abstract]. In: Proceedings of the AACR Special Conference in Cancer Research: Artificial Intelligence and Machine Learning; 2025 Jul 10-12; Montreal, QC, Canada. Philadelphia (PA): AACR; Clin Cancer Res 2025;31(13_Suppl):Abstract nr B042.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.111
GPT teacher head0.440
Teacher spread0.329 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes2
Has abstractyes

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