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Record W4413104696 · doi:10.1093/brain/awaf267

Medium-chain triglycerides improve cognition and systemic metabolism in mouse models of Alzheimer’s disease

2025· article· en· W4413104696 on OpenAlexafffund
Paule E. H. M’Bra, Laura K. Hamilton, Gaël Moquin‐Beaudry, C. Mangahas, Federico Pratesi, Anne Castonguay, Sophia Mailloux, Manon Galoppin, Jessica Avila Lopez, Megan Bernier, Marta Turri, Marian Mayhue, Anne Aumont, M.H. Tetreault, Stephen C. Cunnane, Karl J. L. Fernandes

Bibliographic record

VenueBrain · 2025
Typearticle
Languageen
FieldMedicine
TopicAdipose Tissue and Metabolism
Canadian institutionsUniversité de MontréalCentre Hospitalier Universitaire de SherbrookeUniversité de Sherbrooke
FundersFonds de Recherche du Québec - SantéCanadian Institutes of Health ResearchCourtois FoundationCompute CanadaUniversité de MontréalNatural Sciences and Engineering Research Council of CanadaUniversité de Sherbrooke
KeywordsCognitionDiseaseAlzheimer's diseaseMetabolismNeuroscienceMedicineEndocrinologyInternal medicinePsychology

Abstract

fetched live from OpenAlex

Lifestyle-based interventions, including dietary modifications, can reduce dementia risk. In this regard, dietary supplementation with medium-chain triglycerides (MCT) has shown potential therapeutic benefits in individuals with Alzheimer's disease. These effects are widely presumed to be mediated by hepatic conversion of MCT into circulating ketones. However, the physiological and cellular mechanisms underlying the benefits of MCT remain understudied, particularly in the context of Alzheimer's disease. Here, we investigated the cellular and molecular changes occurring in the brain and systemically in response to dietary supplementation with MCT versus a ketogenic diet. The experimental design consisted of comparing a 70% carbohydrate control diet to either a control diet supplemented with 10% MCT or a carbohydrate-free high-fat ketogenic diet. Diets were tested in two Alzheimer's disease mouse models, slow-progressing 3xTg-AD mice that model pre-symptomatic/early stages and rapidly progressing 5xFAD mice that model late stages of the disease. We found that MCT supplementation and a ketogenic diet both improved hippocampal-dependent spatial learning and memory, increased dendritic spine density of hippocampal neurons and modulated hippocampal expression of genes associated with mitochondrial functions, synaptic structure and insulin signalling in Alzheimer's disease mouse models. However, unlike the ketogenic diet, MCT supplementation did not elevate circulating ketones, suggesting different mechanisms. Indeed, MCT supplementation enhanced the peripheral insulin response of Alzheimer's disease mice, while the ketogenic diet conversely unveiled their latent metabolic vulnerability, increasing their hyperglycaemia, body weight gain and adiposity. The systemic metabolic disturbances of Alzheimer's disease mice correlated with transcriptomic alterations in hepatic lipid metabolism and ketogenesis genes and increased lipid droplet accumulation. These liver metabolic abnormalities were partially reversed by both MCT supplementation and the ketogenic diet, but in distinct ways. Notably, the ketogenic diet selectively triggered hepatic neutral lipid depletion and prominent proinflammatory gene expression, while MCT downregulated expression of cholesterol-related genes. Collectively, these findings reveal that MCT supplementation in the context of Alzheimer's disease improves cognition and systemic metabolism without elevating circulating ketone levels.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.185
Threshold uncertainty score0.535

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.021
GPT teacher head0.283
Teacher spread0.262 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations4
Published2025
Admission routes2
Has abstractyes

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