MétaCan
Menu
Back to cohort
Record W4413821186 · doi:10.1016/j.nbd.2025.107072

Spontaneous pathology in PS19 tauopathy mice progresses via brain networks

2025· article· en· W4413821186 on OpenAlexaff
Denise M. O. Ramirez, Nikhil Bhagwat, Talitha L. Thomas, Apoorva D. Ajay, Ariana Nawaby, Benoı̂t Delatour, Sylvie Bay, Pierre Lafaye, Julie A. Harris, Julian P. Meeks, Marc I. Diamond

Bibliographic record

VenueNeurobiology of Disease · 2025
Typearticle
Languageen
FieldMedicine
TopicAlzheimer's disease research and treatments
Canadian institutionsMontreal Neurological Institute and Hospital
FundersNational Institute of Neurological Disorders and StrokeNational Institutes of HealthRainwater Charitable FoundationNational Institute on AgingAllen InstituteUniversity of Texas Southwestern Medical Center
KeywordsTauopathyNeuroscienceBiologyCognitive sciencePsychologyNeurodegenerationPathologyMedicineDisease

Abstract

fetched live from OpenAlex

Tauopathies are progressive neurodegenerative diseases characterized by cellular accumulation of the microtubule-associated protein tau. Evidence suggests tau is a prion, propagating pathology across brain networks via unique transmissible assemblies which mediate distinct neuropathologies in model systems. Neuroimaging has identified network alterations reflecting distinct patterns of brain atrophy in tauopathy patients. Preclinical studies confirmed transmission of pathological tau between connected brain areas, but relied on inoculation of pathogenic tau protein, leaving a gap in experimental evidence that spontaneous tau aggregates act as prions. We used anti-phospho-tau nanobodies in combination with serial two-photon tomography to immunostain and image whole brains from male and female PS19 mice, which have pan-neuronal expression of full-length human tau containing the P301S mutation. We analyzed patterns of phospho-tau deposition across established brain networks at multiple ages, testing the relationship between structural connectivity and patterns of progressive pathology. We identified core regions with early phospho-tau deposition, and used network propagation modeling to determine the link between tau pathology and connectivity strength. We found that tauopathy progression correlated with structural connectivity, consistent with the prion model. Spontaneous tau propagation was biased in the retrograde direction. These data suggest that despite widespread pathological human tau expression in PS19 mice, spontaneous phospho-tau pathology initiates and propagates along specific brain networks. This work establishes new preclinical methods for studying tau accumulation and propagation, and fills a major gap in our understanding of spontaneous tauopathy. Our novel approach establishes a fundamental role for brain networks in tau propagation, with implications for human disease. Significance statement Our novel methodology for whole brain imaging of p-tau deposition reveals retrograde-dominant network propagation in a tauopathy mouse model. This work establishes new preclinical methods for studying tau accumulation and propagation, and fills a major gap in our understanding of spontaneous tauopathy. Our results establish a fundamental role for brain networks in tau propagation, with implications for human disease.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.294
Teacher spread0.285 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2025
Admission routes1
Has abstractyes

Explore more

Same venueNeurobiology of DiseaseSame topicAlzheimer's disease research and treatmentsFrench-language works237,207