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Record W4414029069 · doi:10.1101/2025.09.01.673570

Inhibited oligodendrogenesis, but not repeated mild traumatic brain injury, impairs attention in adult mice

2025· preprint· en· W4414029069 on OpenAlexafffund
Lisa M. Gazdzinski, Jordan Mak, Paul Fletcher, Anne L. Wheeler

Bibliographic record

VenuebioRxiv (Cold Spring Harbor Laboratory) · 2025
Typepreprint
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMitochondrial Function and Pathology
Canadian institutionsCentre for Addiction and Mental HealthSickKids FoundationHospital for Sick Children
FundersCanadian Institutes of Health Research
KeywordsTraumatic brain injuryNeurosciencePsychologyMedicinePhysical medicine and rehabilitationPsychiatry

Abstract

fetched live from OpenAlex

Abstract Attention problems are among the most common long-lasting cognitive symptoms of mild traumatic brain injury (mTBI) and as attention is fundamental to many aspects of cognition, the effects of attentional impairment can be broad. The brain’s white matter is particularly vulnerable to damage during mTBI. Damage to oligodendrocytes and myelin contributes to cognitive deficits following injury and myelin plasticity is a potential mechanism for functional recovery. The aim of this work was to assess attentional impairment following mTBI in mice and evaluate the role of newly generated oligodendrocytes in recovery. This study used the Myrf conditional knockout mouse model, in which the Myrf gene, required for oligodendrocyte precursor cell (OPC) differentiation into mature myelinating oligodendrocytes, is deleted from OPCs following tamoxifen injection, thereby halting oligodendrogenesis. Mice were trained on the 5-Choice Serial Reaction Time (5-CSRT) task before receiving tamoxifen followed by three mTBI or sham procedures. Attention was probed on the 5-CSRT with decreasing stimulus durations at four time points following injury out to 12 weeks. While no attentional impairment was observed in mice with mTBI, OPC-MyrfKO mice showed lower choice accuracy (β = −2.85%, p = 0.03) and more omitted trials (β = 4.09%, p = 0.02) across injury groups, time points and stimulus durations, suggesting that active oligodendrogenesis is required for sustained attention. These results suggest that this mouse model of mTBI was not severe enough to impact attention as measured by the 5-CSRT task, however myelin plasticity in adulthood may contribute to attention and complex task performance. Significance Statement Attention deficits are common after mild traumatic brain injury (mTBI), yet therapeutic targets remain unclear. This study investigated the role of adult myelin plasticity in cognitive recovery by using a mouse model that blocks new myelin formation. While mild injury alone did not impair attention, mice unable to generate new myelin showed reduced response accuracy. These findings suggest that adult oligodendrogenesis supports attention. Understanding how myelin plasticity influences behaviour could reveal new targets for enhancing recovery after brain injury, highlighting the importance of supporting myelin health in therapeutic strategies.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.012

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.000
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0010.000
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.247
Teacher spread0.232 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes2
Has abstractyes

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