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Record W4414465167 · doi:10.1158/2326-6074.cimm25-b022

Abstract B022: Investigating Biased GPCRs Activation by Nutraceutical Caffeine and Nicotine in Transactivating Glycosylated Receptors to Induce EMT in Cancer Metastasis

2025· article· en· W4414465167 on OpenAlexaffabout
Yunfan Li, Myron R. Szewczuk

Bibliographic record

VenueCancer Immunology Research · 2025
Typearticle
Languageen
FieldMedicine
TopicCancer, Stress, Anesthesia, and Immune Response
Canadian institutionsQueen's University
Fundersnot available
KeywordsG protein-coupled receptorReceptorCancer cellSignal transductionInflammationCaffeineCell signaling

Abstract

fetched live from OpenAlex

Abstract Chronic inflammation plays a central role in tumor initiation, progression, and metastasis. G protein-coupled receptors (GPCRs) are critical regulators of inflammation and cancer-associated signaling. Previous work from our lab revealed that GPCRs form functional heteromeric complexes with neuraminidase-1 (Neu1) and matrix metalloproteinase-9 (MMP9), which transactivate glycosylated receptor tyrosine kinases (RTKs) and Toll-like receptors (TLRs), promoting NF-κB-mediated epithelial–mesenchymal transition (EMT). This research explores how two widely consumed nutraceutical compounds, caffeine and nicotine, act as biased agonists at GPCRs to modulate this signaling axis in cancer progression. We hypothesize that caffeine and nicotine enhance Neu1–MMP9–GPCR–RTK/TLR interactions, selectively activating pro-inflammatory and oncogenic pathways. To test this, RAW-Blue macrophages and two human cancer cell lines (PANC-1 and MDA-MB-231) were used to assess Neu1 sialidase activity, NF-κB transcriptional activity using a secreted embryonic alkaline phosphatase (SEAP) reporter assay, and co-localization of GPCRs with Neu1 by immunofluorescence microscopy. Additionally, EMT markers, such as E-cadherin and N-cadherin, were evaluated by immunofluorescence staining. Cell migration and metastatic potential were assessed via scratch wound assays and cell tunneling experiments. Our preliminary findings show that nicotine significantly promotes NF-κB activation, EMT marker expression, and cell migration, characterized by increased N-cadherin and decreased E-cadherin. In contrast, caffeine exhibits opposing effects, decreasing NF-κB activation and cell migration, and maintaining epithelial marker expression. These results indicate distinct and opposing roles for these compounds in regulating cancer cell behavior through biased GPCR signaling. In summary, this work highlights novel regulatory mechanisms of Neu1–MMP9–GPCR signaling modulated by common dietary compounds. These findings may support the development of low-cost, accessible therapeutic strategies to mimic or block nutraceutical effects in cancer and inflammation management. Citation Format: Yunfan Li, Myron. R. Szewczuk. Investigating Biased GPCRs Activation by Nutraceutical Caffeine and Nicotine in Transactivating Glycosylated Receptors to Induce EMT in Cancer Metastasis [abstract]. In: Proceedings of the AACR Special Conference in Cancer Research: Mechanisms of Cancer Immunity and Cancer-related Autoimmunity; 2025 Sep 24-27; Montreal, QC, Canada. Philadelphia (PA): AACR; Cancer Immunol Res 2025;13(9 Suppl):Abstract nr B022.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.002
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.306
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0020.002
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.003
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.002
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.092
GPT teacher head0.427
Teacher spread0.336 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes2
Has abstractyes

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