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Record W4414653777 · doi:10.1128/jvi.00588-25

Primary envelopment of Kaposi’s sarcoma-associated herpesvirus at the nucleoplasmic reticulum

2025· article· en· W4414653777 on OpenAlexafffund
Alexa Wilson, Neale D. Ridgway, Craig McCormick

Bibliographic record

VenueJournal of Virology · 2025
Typearticle
Languageen
FieldMedicine
TopicViral-associated cancers and disorders
Canadian institutionsDalhousie University
FundersCanadian Institutes of Health Research
KeywordsInner membraneCapsidCytoplasmNuclear membraneCell nucleusNuclear poreNucleusNuclear transportViral envelope

Abstract

fetched live from OpenAlex

ABSTRACT Herpesvirus egress begins with primary envelopment of newly assembled capsids at the inner nuclear membrane (INM). Primary envelopment has been observed at the peripheral INM as well as nuclear infoldings. Nuclear infoldings from invaginations of the INM are Type-I nucleoplasmic reticulum (NR), whereas infoldings of both INM and outer nuclear membrane (ONM) are Type-II NR. Here, we report that Kaposi’s sarcoma-associated herpesvirus (KSHV) reactivation from latency and lytic cycle progression correlates with increases in both types of NR, but primary envelopment is restricted to peripheral INM and Type-I NR. These Type-I NR structures co-localized with puncta containing CTP:phosphocholine cytidylyltransferase (CCTα), the enzyme that catalyzes the rate-limiting step in phosphatidylcholine (PtdCho) synthesis that drives the de novo membrane biogenesis and membrane curvature required for NR expansion; CCTα recruitment may provide sufficient Type-I NR to facilitate nuclear egress. Despite the concurrent expansion of Type-II NR, primary envelopment involves a mechanism that specifically targets capsids to Type-I NR. Building upon our observation of capsids lacking envelopes in complex higher-order Type-I NR structures, we used polar lipid dyes, CLICK-labeled fluorescent viral genomes, and fluorescent KSHV capsids to track the fate of NR-associated capsids via live cell microscopy. These studies provide evidence for trafficking of NR-associated capsids toward the nuclear periphery and cytoplasm. Taken together, these findings suggest that nuclear egress occurs not only at the nuclear periphery but also at the Type-I NR. IMPORTANCE Herpesvirus capsids assemble in the cell nucleus but are too large to exit via nuclear pores. Instead, they bud into the inner nuclear membrane to acquire a provisional lipid envelope that is shed through fusion with the outer nuclear membrane, delivering the capsid to the cytoplasm for subsequent steps in assembly and egress. These nuclear membranes are dynamic, with the ability to fold into invaginations that access the nuclear interior. Here, we demonstrate that during Kaposi's sarcoma-associated herpesvirus (KSHV) replication, nuclear membrane infolding increases, coinciding with recruitment of a host enzyme required for PtdCho synthesis at these sites. We observed accumulation of KSHV capsids at infoldings of the inner nuclear membrane and tracked the association and trafficking of fluorescent viral particles through these structures by live cell microscopy. This complements a more well-established mechanism of KSHV egress at the nuclear periphery and suggests versatility in nuclear egress mechanisms.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.007
GPT teacher head0.251
Teacher spread0.244 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes2
Has abstractyes

Explore more

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