MétaCan
Menu
Back to cohort
Record W4415024770 · doi:10.1038/s44321-025-00316-1

Glia inflammation and cell death pathways drive disease progression in preclinical and early AD

2025· article· en· W4415024770 on OpenAlexafffund
Marcel S. Woo, Joseph Therriault, Seyyed Ali Hosseini, Yi-Ting Wang, Arthur C. Macedo, Nesrine Rahmouni, Étienne Aumont, Stijn Servaes, Cécile Tissot, Jaime Fernandez‐Arias, Lydia Trudel, Brandon J. Hall, Gleb Bezgin, Kely Quispialaya-Socualaya, Marina Gonçalves, Tevy Chan, Jenna Stevenson, Yansheng Zheng, Robert Hopewell, Ilaria Pola, Kübra Tan, Guglielmo Di Molfetta, Firoza Z Lussier, Gassan Massarweh, Paolo Vitali, Jean‐Paul Soucy, Serge Gauthier, Nicholas J. Ashton, Kaj Blennow, Tharick A. Pascoal, Henrik Zetterberg, Andréa Lessa Benedet, Pedro Rosa‐Neto

Bibliographic record

VenueEMBO Molecular Medicine · 2025
Typearticle
Languageen
FieldNeuroscience
TopicNeuroinflammation and Neurodegeneration Mechanisms
Canadian institutionsMcGill University Health CentreMcGill UniversityMontreal Neurological Institute and Hospital
FundersFonds de Recherche du Québec - SantéFonds de recherche du QuébecOlav Thon StiftelsenKirsten og Freddy Johansens FondUK Dementia Research InstituteCure Alzheimer's FundVetenskapsrådetNational Institute for Health and Care ResearchDeutsche ForschungsgemeinschaftEuropean Partnership on MetrologyCorona-StiftungCanadian Institutes of Health ResearchWeston Brain InstituteStiftelsen för Gamla TjänarinnorFamiljen Erling-Perssons StiftelseFondation Brain CanadaAlzheimer's Association
KeywordsNeuroinflammationNeurodegenerationInflammationMicrogliaProgrammed cell deathDiseaseImmune systemCerebrospinal fluidTraumatic brain injury

Abstract

fetched live from OpenAlex

Accumulation of amyloid-β (Aβ) and neurofibrillary tangles (NFTs) are followed by the activation of glia cells and infiltration of peripheral immune cells that collectively accelerate neurodegeneration in preclinical AD models. Yet, the role of neuroinflammation for neuronal injury and disease progression in preclinical and early symptomatic AD remains elusive. Here, we combined multiplexed immunoassays and SomaScan proteomics of the cerebrospinal fluid (CSF) with MRI and PET brain imaging of people across the AD continuum to identify pathways that are associated with AD progression. Unbiased clustering revealed that glia-mediated inflammation, activation of cell death pathways (CDPs) and synaptic pathologies were among the earliest Aβ-induced changes, and were associated with disease progression in preclinical AD. Mediation analysis revealed that activation of CDPs were decisive drivers of inflammation in early symptomatic AD. The cycle of glia-mediated neuroinflammation and neuronal injury characterizes preclinical AD and has implications for novel treatment approaches.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.025
Threshold uncertainty score0.529

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.027
GPT teacher head0.307
Teacher spread0.280 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2025
Admission routes2
Has abstractyes

Explore more

Same venueEMBO Molecular MedicineSame topicNeuroinflammation and Neurodegeneration MechanismsFrench-language works237,207