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Abstract 4365971: Estrogen receptor alpha inhibits right ventricle cardiomyocyte NLRP3 inflammasome activation and restores right ventricular contractile function in low estrogen states

2025· article· en· W4415793015 on OpenAlexaff
Rafael Sobrano Fais, Erica Das Neves Palotta, Katrina W. Kopf, K.M. Massad, Evandro Neto Neves, Christopher Hoffer, Avram Walts, Andrea L. Frump, Neil M. Goldenberg, Sophie Givens, Alice Bourgeois, Chen-­Shan Chen Woodcock, Irina Petrache, KC Woulfe, Soni Savai Pullamsetti, Olivier Boucherat, Steeve Provencher, Brenda M. Ogle, Sébastien Bonnet, Tim Lahm

Bibliographic record

VenueCirculation · 2025
Typearticle
Languageen
FieldMedicine
TopicPulmonary Hypertension Research and Treatments
Canadian institutionsInstitut universitaire de cardiologie et de pneumologie de QuébecUniversity of Toronto
Fundersnot available
KeywordsColocalizationContractilityEstrogenVentricleEstrogen receptorEstrogen receptor alphaAlpha (finance)

Abstract

fetched live from OpenAlex

Background: The NLRP3 inflammasome is implicated in right ventricular (RV) dysfunction, particularly in low endogenous estrogen states found in males and postmenopausal females. We hypothesized that NLRP3 inhibition through estrogen receptor alpha (ERα) enhances RV contractility and improves RV-pulmonary artery (PA) coupling. Methods: RVs from male and pre- or postmenopausal female PH patients with RV failure (RVF) were assessed for NLRP3 activation by analyzing proteomics data and staining for NLRP3 and ASC. Male or female human induced pluripotent stem cell-derived cardiomyocytes (iPSC-CMs) were treated with endothelin-1 (ET1) ± 17beta-estradiol (E2). RV cardiomyocytes (RVCMs) were isolated from male and intact or ovariectomized female rats treated with monocrotaline (MCT) or pulmonary artery banding (PAB). Male or female ERα loss-of-function mutant (ERαmut) rats were employed to study the effects of ERα. RV-PA coupling was assessed by pressure-volume loops (PV-loops) in the WT and ERαmut MCT-treated rats. NLRP3 activation in RVCMs and iPSC-CMs was assessed by NLRP3-ASC colocalization and downstream targets. RVCM contractility and cytosolic calcium (c-Ca2+) were evaluated via IONOPTIX system. P<0.05 was considered significant. Results: Proteomics and immunofluorescence studies revealed that upregulation of NLRP3 activity and signaling in human RVF are more pronounced in males and postmenopausal females (p<0.05). In ET-1-treated iPSC-CMs, NLRP3 was more activated in male than female iPSC-CMs (p<0.05). RVCMs from male, but not female, MCT- or PAB-rats demonstrated increased NLRP3-ASC colocalization (p<0.05). E2 treatment prevented MCT-induced impairment of RV-PA coupling and had beneficial effects on survival. E2 treatment of male and OVX female wildtype (WT) rat RVCMs reduced NLRP3-ASC co-localization and increased Ca2+-dependent contractility (p<0.05). Similarly, E2 treatment in male iPSC-CMs prevented ET-1-induced NLRP3 activation. E2’s inhibitory effects on NLRP3 activation, impairment of RV-PA coupling, improved survival, NLRP3-induced contractile dysfunction, and c-Ca2+ in male WT rat RVCMs were abrogated in ERαmut rat RVCMs (p<0.05). Conclusion: NLRP3 activation impairs RV contractile function in a sexually dimorphic manner. The E2-ERα pathway protects against this dysfunction. E2 enhances RV function and survival in both male and postmenopausal models of RVF, suggesting its potential as a therapeutic target in low estrogen states.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.010
Threshold uncertainty score0.033

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0100.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.254
Teacher spread0.245 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2025
Admission routes1
Has abstractyes

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