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Abstract C034: Obesity-Linked Leptin Signaling Promotes Immune Checkpoint Expression and Tumor Cell Survival

2025· article· en· W4417201691 on OpenAlexaboutno aff
Iqra Kousar

Bibliographic record

VenueClinical Cancer Research · 2025
Typearticle
Languageen
FieldNeuroscience
TopicRegulation of Appetite and Obesity
Canadian institutionsnot available
Fundersnot available
KeywordsLeptinGene knockdownAdipokineImmune systemImmune checkpointLeptin receptorTumor progressionDownregulation and upregulationGene silencingApoptosis

Abstract

fetched live from OpenAlex

Abstract Background: Obesity promotes cancer progression and immune evasion. B7-H3 (CD276), an immune checkpoint protein, is overexpressed in cancers and linked to tumor growth and therapy resistance. This study investigates whether leptin, an adipokine elevated in obesity, regulates B7-H3 expression and contributes to tumor progression and immune escape. Methods: HeLa and T47D cell lines were used to model tumor cells in the early phase of this study. Cells were treated with different concentrations of leptin to evaluate changes in B7-H3, phosphorylated STAT3 (pSTAT3), and PD-L1 expression. Quantitative real-time PCR (qRT-PCR) and Western blotting were employed to assess mRNA and protein expression, respectively. Leptin receptor (OBRb) knockdown was achieved using siRNA to investigate the involvement of leptin signaling. Apoptosis following OBRb knockdown was measured by flow cytometry using Annexin V/Propidium Iodide staining. Results: Leptin treatment led to significant upregulation of B7-H3, pSTAT3, and PD-L1 at both mRNA and protein levels. Silencing of OBRb reduced their expression and increased apoptotic cell populations, indicating a role for leptin in promoting tumor survival and immune evasion. Conclusion: These preliminary findings suggest leptin contributes to obesity-driven tumor progression via the B7-H3–pSTAT3–PD-L1 axis. Further in-depth studies are needed to validate these mechanisms and explore their potential as therapeutic targets in obesity-associated cancers. References: 1. Getu AA, Tigabu A, Zhou M, Lu J, Fodstad Ø, Tan M. New frontiers in immune checkpoint B7-H3 (CD276) research and drug development. Molecular Cancer. 2023 Mar 2;22(1):43. 2. Picarda E, Galbo Jr PM, Zong H, Rajan MR, Wallenius V, Zheng D, Börgeson E, Singh R, Pessin J, Zang X. The immune checkpoint B7-H3 (CD276) regulates adipocyte progenitor metabolism and obesity development. Science advances. 2022 Apr 27;8(17):eabm7012. Acknowledgements: This research is in part funded by China Medical University Ying-Tsai Scholar Fund CMU109-YT-04 (to M.T.) Citation Format: Iqra Kousar. Obesity-Linked Leptin Signaling Promotes Immune Checkpoint Expression and Tumor Cell Survival [abstract]. In: Proceedings of the AACR Special Conference in Cancer Research: The Rise in Early-Onset Cancers—Knowledge Gaps and Research Opportunities; 2025 Dec 10-13; Montreal, QC, Canada. Philadelphia (PA): AACR; Clin Cancer Res 2025;31(23_Suppl):Abstract nr C034.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.009
Threshold uncertainty score0.030

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0090.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.172
GPT teacher head0.459
Teacher spread0.287 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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