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Record W6977810231 · doi:10.7939/82030

An Investigation into the Expression and Role of TSLP During the Anti-Viral Response of Airway Epithelial Cells

2025· dissertation· en· W6977810231 on OpenAlexaboutno aff

Bibliographic record

VenueUniversity of Alberta Library · 2025
Typedissertation
Languageen
FieldMedicine
TopicDermatology and Skin Diseases
Canadian institutionsnot available
Fundersnot available
KeywordsThymic stromal lymphopoietinImmune systemRespiratory epitheliumEpitheliumMediatorAirwayCytokineRespiratory tractStromal cell

Abstract

fetched live from OpenAlex

The airway epithelium is one of the first lines of defence against airborne pathogens. Airway epithelial cells are crucial in initiating and directing the subsequent immune response to deal with invading pathogens rapidly. An early mediator produced by epithelial cells is the alarmin cytokine, thymic stromal lymphopoietin (TSLP), which is known to activate several immune cells. However, overexpression of TSLP is the underlying cause of several diseases, including asthma and allergy, and contributes to increased pathophysiology of other respiratory viruses. Little is known regarding the role of the airway epithelium and TSLP during SARS- CoV-2 infections, the virus responsible for COVID-19. Additionally, the role of TSLP autocrine/paracrine signalling within the epithelium is poorly understood. We investigated the expression of TSLP in COVID-19 patients at the University of Alberta hospital and found that they had a trend towards elevated systemic TSLP, although this was not statistically significant, which correlated with increased hospitalization duration. Several other cytokines were measured, and significant plasma IL-15 and CXCL10/IP-10 increases were detected. In cultured bronchial epithelial cells from healthy donors, we measured significant increases in intracellular and supernatant TSLP expression in response to SARS-CoV-2 infection but not in nasal or gut epithelial cells. To increase our understanding of the role TSLP has within the epithelium, we created a simple model to virally induce epithelial-derived TSLP production and block it using TSLP- neutralizing antibodies. We confirmed that the viral mimetic poly I:C can induce a strong TSLP response through a TLR3-specific mechanism. Furthermore, we characterized the expression of several key antiviral and inflammatory genes, including TSLP, TSLPR, RIG-I, MDA5, IL-25, IL-33, TL1A) and interferons. For the first time, we observed that TL1A is significantly I upregulated in airway epithelial cells in response to poly I:C treatment. Additionally, we made a novel observation showing that epithelial-derived TSLP may contribute to regulating antiviral receptors such as RIG-I and MDA5. Our goal is that the data presented in this thesis sheds light on the vital role of the airway epithelium and TSLP during COVID-19 infections and the broader context of antiviral defences. Our findings are intended to increase our understanding and appreciation for the important role of epithelial cells during viral infections.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.003
GPT teacher head0.192
Teacher spread0.189 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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