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Record W7001334536

J-domain protein molecular chaperones in inherited cerebellar ataxia: cellular dysfunction and altered metabolism associated with loss of DNAJC29/sacsin and DNAJC3

2024· other· en· W7001334536 on OpenAlexaboutno aff

Bibliographic record

VenueQueen Mary Research Online (Queen Mary University of London) · 2024
Typeother
Languageen
Field
Topic
Canadian institutionsnot available
Fundersnot available
KeywordsPhenotypeAtaxiaChaperone (clinical)Heat shock proteinCellular modelSpinocerebellar ataxiaMutationLoss functionCellular stress responseProtein aggregationCellular compartment
DOInot available

Abstract

fetched live from OpenAlex

Proteins containing a J-domain, (known as DnaJ proteins) act to recruit the Hsp70 molecular chaperone machinery to multiple cellular processes. Mutations in several J-domain proteins cause neurological disorders, that are considered “chaperonopathies” as the molecular pathology is believed to be associated with loss of chaperone activity. This thesis focuses on two of these conditions that have cerebellar ataxia as a core component of their phenotype: Autosomal Recessive Spastic Ataxia of Charlevoix-Saguenay (ARSACS), caused by mutations in the SACS gene, which encodes DNAJC29/sacsin; and an ataxia syndrome that also presents with early-onset diabetes mellitus, caused by mutations in DNAJC3. The overarching aim of this thesis was to better understand the cellular consequences of sacsin and DNAJC3 dysfunction, with a focus on metabolic pathways. The first part of the thesis investigated alterations in cellular metabolism in a neuronal cell model of ARSACS. This identified metabolic rewiring as a consequence of sacsin knockout and highlighted potential targets for therapeutic interventions, such as modulation of GABA neurotransmitter levels. Further investigation of the cellular phenotype associated with loss of sacsin, characterised disruption of the microtubule cytoskeleton and its regulators (e.g., tau), altered integrin trafficking and impaired focal adhesion dynamics. These aspects of the loss of sacsin phenotype were further investigated in the mouse retina, which was selected based on the presence of a retinal phenotype in ARSACS patients. In the final part of the thesis, a novel cellular model for DNAJC3 loss of function was developed. Molecular characterisations of this model were then performed by metabolomic and transcriptomic analysis. In summary, defining cellular deficits associated with the loss of sacsin and DNAJC3 is a valuable first step towards understanding molecular mechanism of disease and identification of potential therapeutic strategies for these ataxias. It also broadens our understanding of the cellular roles of J-domain proteins.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow), Research integrity
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.741
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0020.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0020.000
Bibliometrics0.0040.003
Science and technology studies0.0000.002
Scholarly communication0.0000.000
Open science0.0010.001
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.017
GPT teacher head0.249
Teacher spread0.232 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designNot applicable
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

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