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Record W7011661993

Nuclear exclusion of AID limits off target activity by enforcing productive targeting

2018· dissertation· en· W7011661993 on OpenAlexafffund

Bibliographic record

VenueeScholarship@McGill (McGill) · 2018
Typedissertation
Languageen
FieldImmunology and Microbiology
TopicT-cell and B-cell Immunology
Canadian institutionsMcGill University
FundersNational Cancer InstituteFonds de Recherche du Québec - SantéNatural Sciences and Engineering Research Council of CanadaCanadian Institutes of Health ResearchCancer Research Society
KeywordsSomatic hypermutationCytidine deaminasePoint mutationMutationDNAMutantMutagenesisChromatin
DOInot available

Abstract

fetched live from OpenAlex

Activation induced deaminase (AID) is an enzyme that deaminates deoxycitidine to deoxyuridine on single stranded DNA.AID expression is mostly restricted to antigen activated B-lymphocytes, wherein its activity is critical for both somatic hypermutation (SHM) and class-switch recombination (CSR), which underpin secondary antibody diversification.This is due to the targeted activity of AID at the immunoglobulin (Ig) genes, which encode for the antibody genes.DNA deamination catalyzed by AID initiates the generation of single point mutations for SHM, and double stranded breaks for CSR, both potentially harmful lesions.Importantly, multiple mechanisms regulate AID in order to limit its off-target activity, outside the Ig loci, which has been found to be potentially oncogenic and cytotoxic.For my thesis, I used structure-function studies of AID in order to better understand post-translational regulation of its activity.In a first chapter, evolutionary comparison and structure-function analyses revealed that a structural conformation of a C-terminal domain of AID was required for its association to eEF1A1, which mediated the retention of AID in the cytoplasm.Mutations that disrupted AID cytoplasmic retention, or pharmacological inhibition of eEF1A1, caused AID to accumulate in the nucleus and to generate more mutations on-and off-target.This demonstrated that eEF1A1-dependent retention of AID in the cytoplasm was necessary to restrict its activity.In a second chapter, a structure-function study of AID identified a domain dispensable for its catalytic activity, but critical for SHM and CSR activity in B cells.iii Mutants of this domain were able to functionally distinguish the recruitment of AID to the chromatin from its mutagenic activity in B cells.This revealed that at the chromatin AID requires a licensing step in order to associate with the elongating transcription machinery, and become mutagenic.Interestingly, this licensing is bypassed when AID is forced to accumulate in the nucleus.In a third chapter, studies of the C-terminal region of AID further clarified its role during CSR.This study identified a structural requirement of the C-terminus during CSR, downstream from DNA deamination.This study supports a role for AID, via the Cterminus, to recruit other factors during CSR, likely to mediate proper DNA repair.All together, these studies highlight the intricate regulation of AID activity in B cells.They also suggest that preventing accumulation of AID in the nucleus limits offtarget activity by enforcing a regulatory licensing step at the chromatin.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.224
Teacher spread0.214 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes2
Has abstractyes

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