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Record W7042754551

The role of semaphorin 4C in B cells during allergic airways disease

2017· dissertation· en· W7042754551 on OpenAlexaff

Bibliographic record

VenueeScholarship@McGill (McGill) · 2017
Typedissertation
Languageen
FieldNeuroscience
TopicAxon Guidance and Neuronal Signaling
Canadian institutionsMcGill University
FundersUniversity of Otago
KeywordsImmune systemSemaphorinImmunoglobulin ECytokineInterleukin 4AntibodyImmunoglobulin class switchingReceptorInterleukin-1 receptor
DOInot available

Abstract

fetched live from OpenAlex

Allergic airways disease (AAD) is an inflammatory disorder of the airways that is associated with airway hyperresponsiveness (AHR) and airway remodeling.It is characterized by narrowed airways with marked pulmonary eosinophilia, elevated Th2 cytokines and increased peripheral IgE.IgE-producing cells -B lymphocytes -are considered important for AAD progression.Upon allergen exposure, B cells undergo IgE class switching in the presence of Th2 cytokines IL-4 and IL-13.Our laboratory detected a significant increase in Semaphorin 4C (Sema4C) mRNA levels in isolated human tonsillar B cells that were stimulated with anti-CD40 antibodies and IL-4r or L-13.Class 4 semaphorins are of particular importance in immune responses, however, the function of Sema4C in the immune system has never been studied.Our aim was to delineate the function of Sema4C in B cells during Th2 immune responses.Immunofluorescent staining revealed that Sema4C expression was induced by Th2 cytokine stimulation on human activated memory B cells.Sema4C co-localized with its binding partner, Plexin B2, at a synapse-like pole of activated B cells.Sema4c-deficient (Sema4c -/-) B cells failed to polarize and form a synapse-like structure when stimulated in vitro.The decreased polarization was associated with decreased B cell receptor (BCR) signaling and impaired immunoglobulin production.These results suggest that Sema4C is required for polarization and potentialy immune synapse formation, which contributes to B cell differentiation.Using a murine ovalbumin (OVA)-driven AAD model, we determined using both Sema4C deficient mice (Sema4C -/-) as well as chimeric mice with Sema4C deleted specifically in B cells (S4C-B mice) demonstrated exacerbated pulmonary eosinophilia, elevated IL-4 and IL-5 in iv bronchial alveolar lavage fluid (BAL), and increased peripheral OVA-specific IgE.Further analysis showed that in CD138 + B cells, Sema4c -/-B cells exhibited increased IL-4 and decreased IL-10 production, as compared to wild-type (WT) B cells.Transferring Sema4c -/-CD19 + CD138 + cells to WT recipients reproduced the increased Th2 inflammation in S4C-B mice.We also showed that transfer of WT CD19 + CD138 + IL-10 + cells to Sema4C deficient mice suppressed Th2 inflammation in the antigen-driven AAD model.We therefore concluded that Sema4C is required for the differentiation of a novel regulatory B cell population, CD19 + CD138 + IL-10 + , in AAD.In summary, we identified Sema4C as a critical protein for polarization and immune synapse formation in activated B cells.It is required for regulatory cytokine production from CD19 + CD138 + cells.Our studies highlight the importance of B-lymphocytes as regulatory cells in allergic inflammation, as well as providing evidence that regulatory B cell development requires functional immune synapse formation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.017
GPT teacher head0.244
Teacher spread0.227 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2017
Admission routes1
Has abstractyes

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