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Record W7064672589

Characterizing the role of mitochondrial dysfunction in Autosomal Recessive Spastic Ataxia of Charlevoix- Saguenay (ARSACS)

2016· dissertation· en· W7064672589 on OpenAlexaffabout

Bibliographic record

VenueeScholarship@McGill (McGill) · 2016
Typedissertation
Languageen
FieldPhysics and Astronomy
TopicParticle Detector Development and Performance
Canadian institutionsMcGill UniversityMcGill University Health Centre
Fundersnot available
KeywordsMitochondrionAtaxiaIntermediate filamentNeurofilamentCytoskeletonClimbing fiberFibroblastNeurodegenerationVimentin
DOInot available

Abstract

fetched live from OpenAlex

Autosomal Recessive Cerebellar Ataxia of Charlevoix-Saguenay (ARSACS) is a complex inherited neurodegenerative disorder. First described in Quebec in 1978, ARSACS is caused by mutations in the SACS gene that encodes the giant protein of 4579 amino acids, Sacsin. Sacsin has been localized to mitochondria and the cytoplasm, but its function is unknown, and as a result the pathological mechanisms of this complex disease are currently poorly understood. Two features of ARSACS are intermediate filament bundles and elongated mitochondria. The purpose of this project was to evaluate mitochondrial function and to test the hypothesis that mitochondrial abnormalities in ARSACS are a downstream consequence of disordered intermediate filaments. To this end, two cellular models were used: motor neurons in Sacs-/- mouse-derived spinal cord-dorsal root ganglion (DRG) cultures and cultured skin fibroblasts derived from patients. Neurofilament bundling and a modest, but statistically significant, increase in length of mitochondria in motor neuron dendrites were observed after three weeks in vitro. This increase in mitochondrial length was more evident in fibroblasts cultured from ARSACS patients. Human fibroblasts also demonstrated similar bundling of vimentin intermediate filaments, validating the relevance of this model to the neuronal phenotype. With regard to interrogation of mitochondrial function, BN-PAGE of fibroblast mitochondria determined no difference in respiratory chain complex abundance or assembly, and bromouridine incorporation demonstrated no significant change in localization of newly synthesized mitochondrial RNA to RNA granules. Additionally, this study evaluated an intermediate filament-related phenotype that encompasses proteins involved in anchoring of the cytoskeleton to the nucleus (nesprin), consistent with displacement of organelles caused by bundling of intermediate filaments. Finally, to further examine the relationship between intermediate filament and mitochondrial phenotypes in ARSACS, these parameters were measured in fibroblasts from patients with Giant Axonal Neuropathy, a generalized disorder of intermediate filament organization. While intermediate filament bundling and displacement of nesprin and endoplasmic reticulum were found in both ARSACS and GAN fibroblasts, the latter cell line exhibited no change in mitochondrial length. This study provides evidence that changes in mitochondrial morphology observed in ARSACS are not a direct secondary effect that arises from the collapse of the intermediate filament network.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.125
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.001
Open science0.0010.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.219
Teacher spread0.210 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2016
Admission routes2
Has abstractyes

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