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Record W7116044477 · doi:10.14740/wjon2610

E2F5 Overexpression in Laryngeal Squamous Cell Carcinoma: Associations With Neutrophil Extracellular Traps in the Tumor Microenvironment

2025· article· en· W7116044477 on OpenAlexvenueno aff

Bibliographic record

VenueWorld Journal of Oncology · 2025
Typearticle
Languageen
FieldImmunology and Microbiology
TopicNeutrophil, Myeloperoxidase and Oxidative Mechanisms
Canadian institutionsnot available
Fundersnot available
KeywordsTumor microenvironmentImmune escapeImmune systemEvasion (ethics)ExtracellularNeutrophil extracellular traps

Abstract

fetched live from OpenAlex

Background: Laryngeal squamous cell carcinoma (LSCC) is a common malignant tumor of the head and neck, associated with smoking and excessive alcohol consumption. The objective was to investigate the expression pattern of E2F transcription factor 5 (E2F5) in LSCC and its association with neutrophil extracellular traps (NETs), elucidating its role in the tumor microenvironment. Methods: At the cellular level, single-cell RNA sequencing (scRNA-seq) was employed to analyze the expression of E2F5 and NETs-related genes (S100A8, S100A9, LCN2, etc.). At the tissue level, spatial transcriptomics (ST) was used to examine the E2F5 expression pattern. At the mRNA level, E2F5 expression was assessed through mRNA expression profiling, and at the protein level, expression was validated using immunohistochemistry (IHC) on tissue specimens, including 10 LSCC cases (laryngeal, hypopharyngeal, and oropharyngeal squamous cell carcinomas) and 10 non-LSCC controls (benign lesions such as mucoceles, hemangiomas, and polyps). Clustered regularly interspaced short palindromic repeats (CRISPR) knockout screening combined with the CERES algorithm was utilized to evaluate the impact of E2F5 on LSCC cell line proliferation, with negative/positive dependency scores indicating suppression/promotion of growth, respectively. Single-sample Gene Set Enrichment Analysis (ssGSEA) was used to analyze the correlation between E2F5 and immune cells, and chromatin immunoprecipitation sequencing (ChIP-seq) was performed to validate the transcriptional regulation of NETs-related genes by E2F5. Statistical analyses included Wilcoxon, standardized mean difference (SMD), receiver operating characteristic (ROC), and summary receiver operating characteristic (sROC). Results: , ROC AUC = 1). Knockdown of E2F5 significantly inhibited proliferation in LSCC cell lines (e.g., BICR31, BICR16) (inhibition score < 0). High E2F5 expression was positively correlated with T-helper cells and natural killer (NK) CD56bright cells (R = 0.251, 0.175, P < 0.05) and negatively correlated with neutrophils and Th17 cells (R = -0.293, -0.260, P < 0.05). Cellular and tissue-level analyses revealed high NETs expression in LSCC, with E2F5 also highly expressed in NETs-related cells and regions. ChIP-seq analysis confirmed that E2F5 regulates NETs-related genes. Functional enrichment analysis indicated that E2F5-related genes are involved in transcriptional regulation, chromatin organization, and immune regulation. Conclusion: E2F5 is highly expressed in LSCC and is associated with the regulation of NETs-related genes. It may contribute to tumor proliferation and immune evasion by reshaping the tumor microenvironment, highlighting E2F5 as a potential therapeutic target that warrants further functional validation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.011
GPT teacher head0.239
Teacher spread0.228 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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