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Record W7116909846 · doi:10.1002/alz70855_098607

A human iPSC‐derived neurosphere model to study functional and transcriptomic properties of microglial neuroprotection in AD

2025· article· en· W7116909846 on OpenAlexaff
Stefan Wendt, Ada J. Lin, Sarah N. Ebert, Declan J Brennan, Wenji Cai, Yanyang Bai, Da Young Kong, Stefano Sorrentino, Christopher J. Groten, Christopher Lee, Jonathan Frew, Hyun B. Choi, Konstantina Karamboulas, Mathias Delhaye, David R Kaplan, Brian A. MacVicar, Haakon B. Nygaard

Bibliographic record

VenueAlzheimer s & Dementia · 2025
Typearticle
Languageen
FieldNeuroscience
TopicNeuroinflammation and Neurodegeneration Mechanisms
Canadian institutionsUniversity of British Columbia HospitalOpal-Rt Technologies (Canada)University of British Columbia
Fundersnot available
KeywordsMicrogliaNeuroprotectionTranscriptomeNeurosphereNeuroinflammationPhenotype

Abstract

fetched live from OpenAlex

BACKGROUND: Alzheimer's disease is characterized by the accumulation of Aβ plaques in the brain, but the pathways leading from early Aβ deposits to eventual neurodegeneration are not fully understood. The specific role of microglia in AD is controversial, with microglial activation to be protective or detrimental depending on disease progression. We developed a 3D human iPSC-derived neurosphere model to better characterize how microglia respond to Aβ pathology. METHOD: Neurospheres were grown from iPSC-derived neuronal progenitor cells and cultured for 60 days. iPSC-derived microglia were added separately to assess their functional impact. Continuous synthetic oligomeric Aβ treatment was used to model amyloidosis. Genetically encoded sensors roGFP1 and GCaMP6f allowed to monitor neuronal oxidative stress and calcium activity, respectively. Single nuclei RNA sequencing (snRNA-seq) was performed to examine transcriptomic differences induced by Aβ and microglia. RESULT: Neurospheres grew to consist of a complex network of healthy mature neurons and astrocytes. The added microglia readily infiltrate the 3D tissue. Chronic Aβ treatment resulted in the formation of plaque-like aggregates and subsequently led to severe oxidative stress and loss of neuronal activity. Microglia were able to prevent these neurotoxic effects in neurospheres that received mild 3 weeks Aβ treatment, but failed to do so after severe 5 weeks treatment. snRNA-seq revealed that, after 3 weeks treatment, microglia induced upregulation of genes linked to oxidative regulation in astrocytes. After 5 week treatment, microglia more noticeably drove the expression of many AD-associated genes, such as APOE, CLU, and FTL, in astrocytes and neurons. Microglia also appear to have enhanced ability for Aβ clearance after treatment with anti-Aβ antibodies. CONCLUSION: We developed a human 3D neurosphere model to study microglial responses to Aβ pathology. In this model, microglia acquire a neuroprotective phenotype which may be enhanced by anti-Aβ antibodies. Microglia are essential for driving the transcriptomic profile linked to AD after severe Aβ treatment. By mimicking critical AD pathological features, this model offers a platform to identify therapeutic targets with translational relevance.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.067
GPT teacher head0.270
Teacher spread0.203 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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