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Record W7116962377 · doi:10.1002/alz70855_098200

The impact of blood‐brain barrier modulation by focused ultrasound on oligodendrogenesis for Alzheimer's disease

2025· article· en· W7116962377 on OpenAlexaff
Kate Noseworthy, Joseph Silburt, Laura M. Vecchio, Alessia Apa, Sheng‐Kai Wu, Luis Fernando Rubio‐Atonal, Brandy Laurette, Kullervo Hynynen, Isabelle Aubert

Bibliographic record

VenueAlzheimer s & Dementia · 2025
Typearticle
Languageen
FieldEngineering
TopicUltrasound and Hyperthermia Applications
Canadian institutionsSunnybrook Health Science CentreSunnybrook HospitalUniversity of Toronto
Fundersnot available
KeywordsContext (archaeology)DiseaseProcess (computing)Modality (human–computer interaction)Focused ultrasound

Abstract

fetched live from OpenAlex

BACKGROUND: Treatments for supporting oligodendrocytes and restoring myelin in Alzheimer disease (AD) are limited. Focused ultrasound (FUS) with microbubbles (MB) increases the permeability of the blood-brain-barrier (BBB) in a controlled, localized and non-invasive manner. We have demonstrated that FUS promotes elements of brain regeneration, including hippocampal neurogenesis. To further explore the regenerative effects of FUS as a therapeutic strategy for AD, we evaluated its impact on oligodendrogenesis. METHOD: TgCRND8 mice were used as a model of amyloidosis. Non-transgenic littermates and C57BL/6J mice were used as controls. FUS was unilaterally targeted to the hippocampus. Coinciding with FUS application, mice received an intravenous injection of MB; MB absorb ultrasound energy and oscillate, increasing the permeability of the BBB for 4 to 24 hours. BrdU was used to label dividing cells. In C57BL/6J mice, we quantified the proliferation of oligodendrocytes precursor cells (OPCs) at various timepoints post-FUS to identify a time course of OPC proliferation. In all strains, oligodendrogenesis was assessed 30D post-FUS. RESULT: In C57BL/6J mice (n = 4M per timepoint), FUS increased the number of OPCs by 6.8-fold in FUS-treated hippocampi compared to untreated hippocampi at 1D post-FUS (mean±SD of 4491±2402 vs. 657±133; p = 0.03) and 2.3-fold at 4D post-FUS (2543±387 vs.1093±221; p = 0.002). This led to a 5.3-fold increase in mature oligodendrocytes (882±222 vs. 166±61; p = 0.003) compared to the untreated hippocampiat 30D. In the presence of amyloid pathology, FUS led to a 1.4-fold increase in mature oligodendrocytes in FUS treated compared to untreated hippocampi (317±139 vs. 231±146; n = 4, 2M, 2F; p = 0.01) at 30D post-FUS. At 7D post-FUS, a trending increase (p = 0.06) in newborn OPCs was observed in FUS-treated (2364±1365; n = 4, 1M, 3F) compared to untreated hippocampi (1455±353). A sample size of 9 mice per group is estimated to reach significance with an expected medium effect size (d=0.5), alpha of 0.05 and a statistical power of 0.80. CONCLUSION: This first-of-its-kind study demonstrates that FUS stimulates oligodendrogenesis in the mouse hippocampus, including in the context of AD pathology. Exploring the process of FUS-induced oligodendrogenesis and gaining mechanistic insights will advance the understanding of FUS as a therapeutic modality for AD and other white matter diseases.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.012
GPT teacher head0.258
Teacher spread0.246 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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