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Record W7117304959 · doi:10.1002/alz70856_102677

Microglia as a key player in Aβ‐related astrocyte reactivity

2025· article· en· W7117304959 on OpenAlexaff
João Pedro Ferrari‐Souza, Guilherme Povala, Nesrine Rahmouni, Bruna Bellaver, Pamela C.L. Ferreira, Douglas Teixeira Leffa, Firoza Z Lussier, Cristiano Schaffer Aguzzoli, Wagner S. Brum, Marco Antônio De Bastiani, Giovanna Carello‐Collar, Wyllians Vendramini Borelli, Joseph Therriault, Arthur C. Macedo, Stijn Servaes, Jenna Stevenson, Ilaria Pola, Serge Gauthier, Diogo O. Souza, Lucas Porcello Schilling, Mychael V. Lourenco, Gallen Triana‐Baltzer, Hartmuth C. Kolb, Andrea L. Benedet, Nicholas J. Ashton, Dana Tudorascu, Henrik Zetterberg, Kaj Blennow, Tharick A. Pascoal, Pedro Rosa‐Neto, Eduardo R. Zimmer

Bibliographic record

VenueAlzheimer s & Dementia · 2025
Typearticle
Languageen
FieldNeuroscience
TopicNeuroinflammation and Neurodegeneration Mechanisms
Canadian institutionsMcGill University
Fundersnot available
KeywordsAstrocyteMicrogliaCrosstalkKey (lock)NeurogliaReactivity (psychology)

Abstract

fetched live from OpenAlex

Abstract Background Glial reactivity has a major role in Alzheimer's disease (AD) etiology and progression, with astrocytes and microglia orchestrating neuroinflammatory responses. Even though experimental evidence indicate that activated microglia can induce astrocyte reactivity, it remains to be elucidated whether microglia activation influences amyloid‐β (Aβ) effects on astrocyte reactivity in the living AD human brain. Using imaging and fluid biomarker data in individuals across the aging and AD clinical spectrum, we tested the hypothesis that microglia influence the effects of Aβ pathology on astrocyte reactivity. Method Data was obtained from the Translational Biomarkers in Aging and Dementia (TRIAD) study. We studied 62 cognitively unimpaired (CU), 26 mild cognitive impairment (MCI), and 13 AD dementia participants who had positron emission tomography (PET) data for TSPO microglial activation ([ 11 C]PBR28) and Aβ plaques ([ 18 F]AZD4694), as well as reactive astrocyte marker plasma glial fibrillary acidic protein (GFAP). We further assessed tau phosphorylation with plasma phosphorylated tau at threonine 217 ( p ‐tau217) and tau aggregation with [ 18 F]MK‐6240 tau tangle PET. Additionally, a subset of 68 CU and 33 MCI individuals from the TRIAD cohort were evaluated with cerebrospinal fluid (CSF) microglial activation marker soluble triggering receptor expressed on myeloid cells 2 (sTREM2), [ 18 F]AZD4694 Aβ PET, and plasma GFAP. Result Regression analyses revealed that Aβ pathology was associated with astrocyte reactivity only in the presence of elevated levels of microglial activation, supporting that microglial activation influences Aβ effects on astrocyte reactivity. Similar results were observed when using TSPO PET and CSF sTREM2 to assess microglial activation (Figure 1). We also found that microglial activation and astrocyte reactivity were jointly associated with tau phosphorylation and aggregation (Figure 2). Importantly, the microglial‐dependent impact of Aβ on astrocyte reactivity contributed to cognitive impairment through tau pathology (Figure 3). Conclusion Our results suggest that microglial activation is a major phenomenon linking Aβ and astrocyte reactivity in the living AD brain. These findings help to elucidate the intricate crosstalk between microglia and astrocytes in the AD brain, offering insights for the development of glia‐targeting therapies.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.276
Teacher spread0.250 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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