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Record W7132989257

Mechanisms Underlying the Modulation of Hepatic Lipid and Lipoprotein Metabolism in Insulin Resistant States: Examining the Contribution of the Gut and Adipose Tissue

2023· dissertation· W7132989257 on OpenAlexfundno aff
Danielle Alvares

Bibliographic record

VenueTSpace · 2023
Typedissertation
Language
FieldMedicine
TopicDrug Transport and Resistance Mechanisms
Canadian institutionsnot available
FundersUniversity of Toronto ScarboroughHeart and Stroke Foundation of CanadaBanting and Best Diabetes Centre, University of TorontoCanadian Institutes of Health ResearchHospital for Sick ChildrenUniversity of Toronto
KeywordsAdipose tissueLipoproteinG protein-coupled bile acid receptorBile acidVery low-density lipoproteinWhite adipose tissueInsulin resistanceLipid metabolismReceptor
DOInot available

Abstract

fetched live from OpenAlex

Dyslipidemia and non-alcoholic fatty liver disease (NAFLD) are complications of insulin resistant states and are primarily caused by alterations in hepatic very low density lipoprotein (VLDL) production. Recent research has implicated a gut-liver-adipose tissue axis involving bile acids and the gut microbiota, as well as adipose tissue secreted hormones and miRNAs in the modulation of hepatic lipid and lipoprotein homeostasis. Bile acid signalling through the Takeda G protein coupled receptor 5 (TGR5) and the gut microbiota have been shown to alter intestinal glucagon-like peptide 1 (GLP-1) production, which has independently been shown to modulate hepatic lipoprotein metabolism. Additionally, adipose tissue expression of neuregulin 4 (Nrg4) has been shown to be altered following BAT activation, and miR-130b has been shown to be altered in obesity. Both Nrg4 and miR-130b been shown to independently modulate hepatic lipoprotein secretion. Therefore, I hypothesize that TGR5 signalling and the gut microbiota exert modulatory effects which may reduce hepatic excursion of VLDL through a GLP1 receptor (GLP-1R) dependent pathway. Further, I propose that alterations in Nrg4 expression observed following BAT activation, and alterations in miR-130b expression in obesity independently modulate hepatic lipid homeostasis. To investigate bile acid mediated regulation of hepatic lipoprotein metabolism, we developed a novel TGR5 knockout (KO) hamster model and characterized the model in normal and insulin resistant states. Subsequently employing both TGR5 KO and GLP-1R KO hamsters, we demonstrated that bile acids can acutely decrease hepatic VLDL secretion, an effect that was dependent on vagal GLP-1R signalling. To assess the involvement of the gut microbiota, we performed antibiotic induced microbiota depletion (AIMD) in hamsters and observed perturbations in hepatic lipid storage and secretion. We next examined the effects of Nrg4 and miR-130b in isolated hepatocytes and in animal models. Both Nrg4 administration and miR-130b overexpression were associated with increased hepatic VLDL secretion. Taken together, data presented in this thesis demonstrates the importance of gut and adipose tissue signalling in the modulation of hepatic lipid accumulation and VLDL production. Modulating these pathways in vivo may be a potential therapeutic approach to correct the dyslipidemia and NAFLD commonly associated with the metabolic syndrome.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.001
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.026
GPT teacher head0.305
Teacher spread0.279 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

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