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Record W7133023117

The Multifaceted Roles of Fragile X Mental Retardation Protein in Developmental Plasticity at a Central Synapse

2021· dissertation· W7133023117 on OpenAlexaff
Ankur Bodalia

Bibliographic record

VenueTSpace · 2021
Typedissertation
Language
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetics and Neurodevelopmental Disorders
Canadian institutionsUniversity of Toronto
Fundersnot available
KeywordsFragile X syndromeSynapseAMPA receptorPostsynaptic potentialFMR1GlutamatergicSilent synapseSynaptic plasticityNMDA receptor
DOInot available

Abstract

fetched live from OpenAlex

During their critical period of sensory development, postsynaptic glutamatergic receptors, namely AMPA receptors (AMPARs) and NMDA receptors (NMDARs), undergo rapid remodeling in abundance and composition to enable fast neurotransmission. Dysregulation of this process can lead to neurodevelopmental disorders such as Fragile X Syndrome (FXS) which shares several mental retardation-associated features of Autism Spectrum Disorders (ASD). This is caused by a loss of Fragile X Mental Retardation Protein (FMRP), a critical translational repressor at the synapse recently implicated in ion channel modulation. To investigate the roles of FMRP in synaptic remodeling of glutamatergic receptors, I have established developmental profiles at the calyx of Held-MNTB synapse in the auditory brainstem by measuring whole-cell electrophysiological synaptic responses from pre-ear canal opening (post-natal day 6-9) and after-hearing onset (post-natal day 16-19) synapses in wild-type and the FMR1-/- mouse model of human FXS. My results indicate the loss of FMRP causes early maturation of AMPARs by accelerating the developmental switch from the slower-gating GluA1-dominant to faster-gating GluA4-dominant subunit composition of AMPARs, consistent with the canonical role of FMRP as an mRNA translational repressor. In contrast, developmental downregulation of NMDARs is not only stunted in FMR1-/- synapses, but also resistant to activity-dependent downregulation. This defect can be acutely rescued by intracellular delivery of the protein binding N-terminus fragment of FMRP during whole-cell patch clamp recordings, suggesting FMRPN-terminal is sufficient for mediating activity-dependent plasticity of NMDARs. Importantly, pulldown of the NMDAR subunit GluN1 and mGluR5 by FMRP in co-immunoprecipitation experiments provides evidence of FMRP binding to mGluR5-NMDAR complexes. This raises the possibility that a protein-protein interaction involving FMRP is engaged in the developmental downregulation of NMDARs. Based on these novel findings, I propose a model whereby activity induced increases in intracellular calcium triggers a two-pronged biochemical cascade through which FMRP differentially regulates two glutamatergic receptors within the same cell type. Altogether, FMRP functions as a canonical translational repressor to regulate AMPAR subunit expression, as well as as a non-canonical signaling molecule to mediate activity-dependent downregulation of NMDARs. These deficits in developmental synaptic remodeling provide mechanistic insights into the sensory hypersensitivity and cognitive impairments in FXS and ASD.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.000
Threshold uncertainty score0.002

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.001
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.254
Teacher spread0.244 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

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