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Record W7133034705

Identification of Novel Regulators of the Hippo Pathway using Positive Selection Genome Wide CRISPR Screens

2022· dissertation· W7133034705 on OpenAlexaff
Sabrina Sen

Bibliographic record

VenueTSpace · 2022
Typedissertation
Language
FieldBiochemistry, Genetics and Molecular Biology
TopicHippo pathway signaling and YAP/TAZ
Canadian institutionsUniversity of Toronto
Fundersnot available
KeywordsHippo signaling pathwaySignal transducing adaptor proteinCRISPRRegulatorPhosphorylationGeneKinaseSignal transductionCellGenetic screen
DOInot available

Abstract

fetched live from OpenAlex

The Hippo pathway is established as a master regulator of tissue growth and organ size and its dysregulation can lead to cancer. The core pathway consists of the kinases MST1/2 and LATS1/2, their adaptor proteins, SAV1 and MOB1A/1B, respectively, and the transcriptional module YAP and TAZ. Multiple upstream signals such as mechanotransduction, GPCRs and metabolic pathways activate MST1/2 which along with SAV phosphorylates and activates LATS1/2 together with MOB proteins which phosphorylate and sequester YAP/TAZ in the cytoplasm leading to their subsequent degradation. The core pathway is well studied, however, many upstream pathway regulators are ill-defined/unknown. To identify novel Hippo pathway regulators, I undertook two positive selection genome-wide CRISPR screens. I generated clonal cell lines of T24 bladder and MDA-MB-231 breast cancer cells expressing two proteins, Cas9 which enabled gene deletion and a FKBPiCaspase9 fusion protein under YAP/TAZ transcriptional control, which when expressed causes cell death upon addition of chemical dimerizer, AP20187. CRISPR-mediated loss of genes promoting YAP/TAZ activity, allows cell survival by downregulating the FKBPiCaspase9 protein. The first screen, in T24 bladder cancer cells identified the phosphatase adaptor protein PPFIA1 as a positive regulator of YAP/TAZ. I demonstrated that loss of PPFIA1 leads to decreased YAP/TAZ protein levels, increased YAP/TAZ phosphorylation, enhanced cytoplasmic localization and iii decreased YAP/TAZ target gene expression. Immunoprecipitation analysis demonstrated that PPFIA1 interacts with YAP. These data suggest a model in which PPFIA1 plays a role in maintaining YAP in a dephosphorylated state, possibly by recruiting a phosphatase. To identify novel regulators that might be relevant to multiple cell lines I performed a second screen in the MDA-MB-231 cells and identified the Gα protein chaperone RIC8A, which also functions as a Guanine Nucleotide Exchange Factor (GEF), as another putative regulator of the Hippo pathway. I showed that abrogation of RIC8A expression mediated a decrease in total YAP /TAZ protein levels, a decrease in YAP /TAZ target gene expression and increased cytoplasmic localization of YAP/TAZ in both bladder and breast cancer cells. Overall, my thesis introduces a novel method to interrogate the Hippo pathway in multiple cancer types and identifies new regulators of YAP and TAZ.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.009

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.016
GPT teacher head0.301
Teacher spread0.285 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2022
Admission routes1
Has abstractyes

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