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Record W7133075226

Signaling Mechanism and Pharmacological Manipulation of Bcl-2 Family Proteins in Apoptosis

2021· dissertation· W7133075226 on OpenAlexafffund
Justin Philip Pogmore

Bibliographic record

VenueTSpace · 2021
Typedissertation
Language
FieldBiochemistry, Genetics and Molecular Biology
TopicCell death mechanisms and regulation
Canadian institutionsUniversity of Toronto
FundersCanadian Institutes of Health ResearchMitacsBundesministerium für Bildung und Forschung
KeywordsBcl-2 familyApoptosisProgrammed cell deathMitochondrionActivator (genetics)Protein familyIntermembrane spaceCellSignal transductionInhibitor of apoptosis
DOInot available

Abstract

fetched live from OpenAlex

Programmed cell death (apoptosis) dysregulation can drive disease pathologies in humans. Too much cell death is an unmet issue in neurodegenerative disease, is associated with decreases in tissue engraftment success, and is a hurdle in biotechnology applications. Conversely, too little cell death is partly associated with the formation of cancer and resistance to chemotherapy treatments. In a single cell, Bcl-2 family proteins orchestrate the life-or-death decision by complex interactions at the mitochondrial outer membrane. Anti-apoptotic proteins like BCL-XL can sequester activator proteins (BID), and pore-forming proteins (BAX and BAK) to keep the cell from undergoing apoptosis. Sensitizer proteins such as BAD, can bind to anti-apoptotic proteins leading to the activation of pore-forming proteins by free activator proteins. When a BAX/BAK pore is formed on the outer membrane of the mitochondria, the cell is committed to apoptosis with no return. Pro-apoptotic factors released from the mitochondrial intermembrane space trigger an apoptotic cascade. Here, I aid in the discovery of a novel inhibition mechanism at the mitochondria where homodimerized BCL-X¬L¬ can also act as the scaffold for a “trigger like” activation of a pro-apoptotic signal. Although Bcl-2 family proteins have been studied for about 40 years, we are still observing novel mechanisms, understanding the function of structural domains, and developing small molecules as future therapies. The first success in clinic to target Bcl-2 family proteins is the anti-apoptotic protein inhibitor, Venetoclax, used to treat chronic lymphocytic leukemia. The purpose of Venetoclax is to repress inhibitory proteins to cause activation of apoptosis in cancer. However, little is known about inhibiting cell death to treat disease. BAX and BAK form pores that commit the cell to death, and by blocking pore formation we can keep cells alive for longer, allowing stressed cells time to recover. Here, I aid in the development of small-molecule BAX/BAK inhibitors to save cells from an apoptotic stimulus. I found that pyrimidine or quinazoline based, FDA-approved kinase inhibitors have low-micromolar affinities for BAX and BAK. With aid from synthetic chemists, I was able to predict chemical changes that removed kinase binding while maintaining the propensity to inhibit BAX and BAK.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.001
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.023
GPT teacher head0.312
Teacher spread0.289 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes2
Has abstractyes

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