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Record W2248508232

Screening of candidate adenovirus expressing shRNAs for functional recovery of dF508-CFTR

2010· article· en· W2248508232 on OpenAlexaboutno aff
Eric J. Sorscher, Jau‐Shyong Hong, Darren M. Hutt, Monica A. Chalfant, DM Roth, William E. Balch, Sabrina Noël

Bibliographic record

VenueDIAL (Catholic University of Leuven) · 2010
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicVirus-based gene therapy research
Canadian institutionsnot available
Fundersnot available
KeywordsΔF508Small hairpin RNATransduction (biophysics)RNA interferenceGene silencingBiologySmall interfering RNAMedicineCell biologyCystic fibrosisMolecular biologyGeneCystic fibrosis transmembrane conductance regulatorGeneticsTransfectionGene knockdownRNABiochemistry
DOInot available

Abstract

fetched live from OpenAlex

Devising molecular strategies that overcome ΔF508 folding and trafficking defects comprise a central objective of CF therapeutic development. An understanding of disease mechanisms can be improved by new method(s) and/or compounds that redirect ΔF508-CFTR to the plasma membrane. RNA interference (siRNA) mediated knock-down of gene expression has proven to be a powerful tool for investigating protein function(s) and advancing drug discovery. BioFocus (a Galapagos company) has developed adenoviral vectors expressing small hairpin RNAs (shRNAs) for genomewide functional screening that allow robust transduction and durable gene repression. In this project, sixty-eight adenoviral-shRNA constructs (targeting 28 high priority genes) were provided by BioFocus to five collaborating laboratories in the United States and Canada. Putative gene targets were identified by a consortium-based review of the existing CF literature. Each research group established independent protocols to investigate effect(s) of gene knock-down (via Ad-shRNA) on ΔF508-CFTR maturation. Protocols included 1) A study of CF bronchial epithelial cells expressing the halide sensitive variant of eYFP and measurement of ΔF508-CFTR activity at the cell surface. 2) Short circuit current in primary human bronchial epithelial cells (ΔF508/ΔF508), 3) Appearance of rescued ΔF508 CFTR at the plasma membrane in CF bronchial epithelial cells monitored biochemically, 4) Effects on CFTR-dependent release of inflammatory markers (chemokines and cytokines) from IB3 cells (ΔF508/W1282X), and 5) Short circuit current and Western blotting in CFBE cells transduced with lentivirus encoding ΔF508-CFTR. Preliminary results indicate significant activity of BioFocus shRNAs in several of the independent protocols and laboratories, particularly against gene products such as AHSA1 and 2 (activators of HSP 90) and HDAC7A (a member of the histone deacetylase family). A profile of shRNAs found to improve ΔF508 processing, including spectrum of activity data and gene-network annotation of the relevant pathways, will be presented. Identifying the most robust molecular targets for ΔF508 CFTR correction (from among hundreds of candidates in the CFTR “interactome”) has been limited by the complexity of the relevant cellular pathways. The studies described here provide a means by which chaperones and other contributors to CFTR misprocessing can be evaluated, prioritized, and better understood in the future for development of new therapeutic approaches and delineating genetic modifiers that contribute to variation in severity of CF onset and disease progression. The project represents a collaboration among members of the CFTR Folding Consortium. Supported by the CFF and NIH.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.023
GPT teacher head0.258
Teacher spread0.235 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2010
Admission routes1
Has abstractyes

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