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Record W2270731020 · doi:10.1007/s00439-015-1616-8

Genetic variation in the immunosuppression pathway genes and breast cancer susceptibility: a pooled analysis of 42,510 cases and 40,577 controls from the Breast Cancer Association Consortium

2015· article· en· W2270731020 on OpenAlexafffund
Jieping Lei, Anja Rudolph, Kirsten B. Moysich, Sabine Behrens, Ellen L. Goode, Manjeet K. Bolla, Joe Dennis, Alison M. Dunning, Douglas F. Easton, Javier Benı́tez, John L. Hopper, Melissa C. Southey, Marjanka K. Schmidt, Annegien Broeks, Peter A. Fasching, Lothar Haeberle, Julian Peto, Isabel dos‐Santos‐Silva, Elinor J. Sawyer, Ian Tomlinson, Barbara Burwinkel, Frederik Marmé, Pascal Guénel, Thérèse Truong, Stig E. Bojesen, Henrik Flyger, Sune F. Nielsen, Børge G. Nordestgaard, Anna González‐Neira, Primitiva Menéndez, Hoda Anton‐Culver, Susan L. Neuhausen, Hermann Brenner, Volker Arndt, Alfons Meindl, Rita K. Schmutzler, Hiltrud Brauch, Ute Hamann, Heli Nevanlinna, Rainer Fagerholm, Thilo Dörk, Natalia Bogdanova, Jaana M. Hartikainen, Laurien Van Dyck, Ann Smeets, Dieter Flesch‐Janys, Ursula Eilber, Paolo Radice, Paolo Peterlongo, Fergus J. Couch, Emily Hallberg, Graham G. Giles, Roger L. Milne, Christopher A. Haiman, Fredrick R. Schumacher, Jacques Simard, Mark S. Goldberg, Vessela Kristensen, Anne‐Lise Børresen‐Dale, Wei Zheng, Alicia Beeghly‐Fadiel, Robert Winqvist, Mervi Grip, Irene L. Andrulis, Gord Glendon, Montserrat García‐Closas, Jonine D. Figueroa, Kamila Czene, Judith S. Brand, Hatef Darabi, Mikael Eriksson, Per Hall, Jingmei Li, Angela Cox, Simon S. Cross, Paul D.P. Pharoah, Mitul Shah, Maria Kabisch, Diana Torres, Anna Jakubowska, Jan Lubiński, Foluso O. Ademuyiwa, Christine B. Ambrosone, Anthony J. Swerdlow, Michael E. Jones, Jenny Chang‐Claude

Bibliographic record

VenueHuman Genetics · 2015
Typearticle
Languageen
FieldMedicine
TopicCancer Immunotherapy and Biomarkers
Canadian institutionsLunenfeld-Tanenbaum Research InstituteMount Sinai HospitalMcGill UniversityUniversity of TorontoRoyal Victoria HospitalUniversité LavalCentre hospitalier universitaire de Québec
FundersNational Cancer InstituteUniversitätsklinikum Hamburg-EppendorfInstituto de Salud Carlos IIICancer Council South AustraliaMedical Research CouncilNational Institutes of HealthAkershus UniversitetssykehusGuy's and St Thomas' NHS Foundation TrustDeutschen Konsortium für Translationale KrebsforschungGesellschaft der Freunde der Medizinischen Hochschule HannoverRheinische Friedrich-Wilhelms-Universität BonnMinistero dello Sviluppo EconomicoAgence Nationale de Sécurité Sanitaire de l’Alimentation, de l’Environnement et du TravailInstitut National Du CancerNational Health and Medical Research CouncilOulun YliopistoDeutsche KrebshilfeMedizinischen Hochschule HannoverNorges ForskningsrådUniversitetet i BergenKuopion Yliopistollinen SairaalaOvarian Cancer Research FundBundesministerium für Bildung und ForschungMinisterio de Economía y CompetitividadStichting Tegen KankerDeutsche Gesetzliche UnfallversicherungNederlandse Organisatie voor Wetenschappelijk OnderzoekKing's College LondonAcademy of FinlandCancer AustraliaAgence Nationale de la RechercheRobert Bosch StiftungYorkshire Cancer ResearchVicHealthNational Breast Cancer FoundationCancer Council Western AustraliaEuropean CommissionFondation du cancer du sein du QuébecBreakthrough Breast CancerNational Institute for Health and Care ResearchSusan G. KomenMcGill University Health CentreUniversity of California, IrvineKreftforeningenDavid F. and Margaret T. Grohne Family FoundationCancer Research UKHamburger KrebsgesellschaftItä-Suomen YliopistoMinistère du Développement Économique, de l’Innovation et de l’ExportationLon V. Smith FoundationEuropean Social FundAgency for Science, Technology and ResearchCancer Council TasmaniaFrancis Crick InstituteCanadian Institutes of Health ResearchSigrid Juséliuksen SäätiöGeneral Secretariat for Research and TechnologyMedisinske fakultet, Universitetet i OsloLigue Contre le CancerDeutsches KrebsforschungszentrumBreast Cancer Research FoundationMcGill UniversityHelsingin ja Uudenmaan SairaanhoitopiiriCancer Council NSWSusan G. Komen for the CureKWF KankerbestrijdingNordic Cancer UnionHerlev HospitalUniversity of Southern CaliforniaAssociazione Italiana per la Ricerca sul CancroBeckman Research Institute, City of HopeNIHR Biomedical Research Centre, Royal Marsden NHS Foundation Trust/Institute of Cancer ResearchFondation de FranceUniversitetet i OsloSundhed og Sygdom, Det Frie ForskningsrådCancer Council VictoriaCalifornia Department of Public HealthHaukeland UniversitetssjukehusU.S. Department of Health and Human ServicesUniversity of Cambridge
KeywordsBiologyBreast cancerHuman geneticsGenetic variationGeneImmunosuppressionCancerGeneticsGenetic associationOncologyBioinformaticsGenotypeSingle-nucleotide polymorphismImmunologyMedicine

Abstract

fetched live from OpenAlex

Immunosuppression plays a pivotal role in assisting tumors to evade immune destruction and promoting tumor development. We hypothesized that genetic variation in the immunosuppression pathway genes may be implicated in breast cancer tumorigenesis. We included 42,510 female breast cancer cases and 40,577 controls of European ancestry from 37 studies in the Breast Cancer Association Consortium (2015) with available genotype data for 3595 single nucleotide polymorphisms (SNPs) in 133 candidate genes. Associations between genotyped SNPs and overall breast cancer risk, and secondarily according to estrogen receptor (ER) status, were assessed using multiple logistic regression models. Gene-level associations were assessed based on principal component analysis. Gene expression analyses were conducted using RNA sequencing level 3 data from The Cancer Genome Atlas for 989 breast tumor samples and 113 matched normal tissue samples. SNP rs1905339 (A>G) in the STAT3 region was associated with an increased breast cancer risk (per allele odds ratio 1.05, 95 % confidence interval 1.03-1.08; p value = 1.4 × 10(-6)). The association did not differ significantly by ER status. On the gene level, in addition to TGFBR2 and CCND1, IL5 and GM-CSF showed the strongest associations with overall breast cancer risk (p value = 1.0 × 10(-3) and 7.0 × 10(-3), respectively). Furthermore, STAT3 and IL5 but not GM-CSF were differentially expressed between breast tumor tissue and normal tissue (p value = 2.5 × 10(-3), 4.5 × 10(-4) and 0.63, respectively). Our data provide evidence that the immunosuppression pathway genes STAT3, IL5, and GM-CSF may be novel susceptibility loci for breast cancer in women of European ancestry.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.113
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.024
GPT teacher head0.289
Teacher spread0.265 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations10
Published2015
Admission routes2
Has abstractyes

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