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The A4-Fla2 flagellin, a dominant antigen in Crohnʼs Disease, is a poor TLR5 agonist

2009· article· en· W2328985452 on OpenAlexaff
Theodore S. Steiner, Sabine Ivison, C Wang, C. J. Elson

Bibliographic record

VenueInflammatory Bowel Diseases · 2009
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicInflammatory Bowel Disease
Canadian institutionsUniversity of British Columbia
FundersNational Institute of Diabetes and Digestive and Kidney DiseasesCrohn's and Colitis Foundation of America
KeywordsTLR5FlagellinImmunologyMicrobiologyAntigenBiologyColitisAntibodyInflammatory bowel diseaseReceptorMedicineToll-like receptorImmune systemInternal medicineDiseaseInnate immune systemBiochemistry

Abstract

fetched live from OpenAlex

Approximately half of patients with Crohn's disease (CD) have measurable serum antibodies to CBir1, a flagellin protein from a nonculturable member of the Lachnospiraceae group of Clostridiales order bacteria. A taxonomically related organism, designated A4, is found in high numbers in the normal mouse colon and expresses a flagellin, Fla2, which also strongly reacts with CD sera. Inflammatory responses to flagellin in the intestinal epithelium are mediated by Toll-like receptor 5 (TLR5) but the role of TLR5 in the generation of pathologic anti-flagellin antibodies in CD remains unknown and has been called into doubt by recent findings that TLR5 is not required for the colitigenic effects of flagellin in the DSS colitis model. It has previously been reported that CBir1 causes cytokine release from hemopoietic cells, but its direct TLR5 agonist activity has never been quantified. To determine if Fla2 and other flagellins that react with CD sera have different TLR5-stimulatory activity than flagellins from Enterobacteriaceae. 1) Recombinant Fla2 and E. coli FliC were used to stimulate HEK 293T cells transiently expressing murine or human TLR5, and IL-8 and CCL20 production were measured. 2) C57Bl/6 mice were injected intraperitoneally with Fla2 or FliC, and serum IL-8 measured after 3h. 3) The fla2 and fliC genes were cloned into a low-copy expression system under control of the native E. coli fliC promoter in E. coli with a chromosomal fliC deletion, and motility was measured. As previously reported, FliC was a potent agonist for both human and murine TLR5 in HEK 293T cells, with an EC50 of 1-5 ng/ml. In contrast, Fla2 was a much weaker agonist, with an EC50 of 100-500 ng/ml for murine TLR5, and greater than 1 μg/ml for human TLR5. This potency difference was also evident in the in vivo model, where IL-6 production was markedly higher in FliC treated mice (4332±1563 pg/ml) than in Fla2-treated mice (111±66 pg/ml). Finally, unlike FliC, Fla2 was unable to confer motility to E. coli:fliC- in the native expression system. Motility of the A4 organism itself was not measured due to the requirement for strict anaerobic conditions. The A4-Fla2 flagellin is a poor agonist for TLR5, with higher activity for murine than human TLR5. This manifests in vivo as a weak inflammatory response to IP injection compared to E. coli FliC. Fla2 also appears to lack the ability to confer motility to E. coli, although the motility of the native A4 strain remains unknown. These results support the hypothesis that TLR5 stimulation and the development of pathological adaptive immune responses in IBD may be inversely correlated.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.234
Teacher spread0.228 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2009
Admission routes1
Has abstractyes

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