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Record W2563552218 · doi:10.1093/biolreprod/85.s1.654

Either Kras Activation or Pten Loss Similarly Enhance the Mutant CTNNB1-Induced Genetic Program to Promote Granulosa Cell Tumor Development in Ovary and Testis.

2011· article· en· W2563552218 on OpenAlexaffabout
Zhilin Liu, Heng‐Yu Fan, Alexandre Boyer, Mayra Tsoi, Marie-Noëlle Laguë, Lisa K. Mullany, Derek Boerboom, JoAnne S. Richards

Bibliographic record

VenueBiology of Reproduction · 2011
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer-related gene regulation
Canadian institutionsUniversité de MontréalCegep de Saint Hyacinthe
Fundersnot available
KeywordsPTENBiologyKRASCancer researchWnt signaling pathwayGranulosa cellOvaryPhenotypeMutationPI3K/AKT/mTOR pathwayCell biologySignal transductionGeneEndocrinologyGenetics

Abstract

fetched live from OpenAlex

The stable activation of the WNT signaling effector beta-catenin (CTNNB1(ex3)) in ovarian granulosa cells results in the formation of premalignant lesions that develop into granulosa cell tumors (GCTs) spontaneously later in life. Although loss of the tumor suppressor gene Pten alone rarely leads to GCTs, its disruption in granulosa cells that concomitantly express a dominant-stable CTNNB1 mutant accelerates tumor formation. Expression of mutant (oncogenic) KRASG12D in granulosa cells results in the arrest of proliferation, differentiation and apoptosis. In this study, we sought to determine if KRAS activation would retard or block the ovarian phenotype of mice expressing dominant-stable CTNNB1. Unexpectedly, transgenic mice bearing mutations of CTNNB1 and KRAS developed early-onset GCTs leading to premature death in a manner similar to the Ctnnb1;Pten mutant mice. GCT cells in these animals were characterized by increased rates of proliferation, decreased apoptosis and reduced expression of markers of granulosa cell differentiation relative to normal ovaries. Microarray and RT-PCR studies were performed comparing control ovaries, ovaries from mice expressing dominant-stable CTNNB1, and ovaries bearing early-stage tumors from mice with the CTNNB1 mutation and either Pten loss or KRAS activation. Results demonstrated that gene regulatory processes induced by CTNNB1 were mostly further enhanced by either KRAS activation or Pten loss, and that the latter mutations largely affected the expression of the same genes in remarkably similar patterns. Granulosa cells from the tumors proliferate in culture and retain expression of selected CTNNB1 target genes. In addition, concomitant activation of CTNNB1 and KRAS in Sertoli cells resulted in the development of granulosa cell tumors of the testis similar to those observed in the Ctnnb1;Pten mutant mice. Real time-PCR studies showed a partial overlap in gene regulatory processes associated with tumor development in the ovarian and testicular GCTs. Collectively, these results suggest that mutant stable CTNNB1 alters the granulosa cell genetic program and predisposes these cells to transformation whereby either KRAS activation or Pten loss induce GCT development from premalignant lesions via highly similar molecular mechanisms. Supported in part by NIH-HD-16229 and HD-07945 (J.S.R.) and the Canadian Institutes of Health Research (D.B.). (poster)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.214
Threshold uncertainty score0.455

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.019
GPT teacher head0.261
Teacher spread0.242 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2011
Admission routes2
Has abstractyes

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