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Record W2589012814 · doi:10.1093/neuonc/now188.231

P10.07 Sialate O-acetylesterase (SIAE) 
over-expression leads to a collapse of the mitochondrial membrane potential and predisposes cells to etoposide treatment in RES256 medulloblastoma cells

2016· article· en· W2589012814 on OpenAlexaff
Rebecca Mather, Katie F. Loveson, Laura Donovan, Geoffrey J. Pilkington, Helen L. Fillmore

Bibliographic record

VenueNeuro-Oncology · 2016
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMitochondrial Function and Pathology
Canadian institutionsSickKids Foundation
Fundersnot available
KeywordsEtoposideMedulloblastomaCell biologyMedicineChemistryBiologyCancer researchMolecular biologyInternal medicineChemotherapy

Abstract

fetched live from OpenAlex

Medulloblastoma is the most common malignant paediatric brain tumour and is the leading cause of childhood cancer related mortality. Current treatment includes maximal safe surgical resection, radiotherapy if the patient is over 3 years of age, and high-dose chemotherapy that usually includes methotrexate, cisplatin, etoposide, vincristine, lomustine and cyclophosphamide. Survivors often suffer significant treatment-related sequelae including endocrine disorders and neurocognitive deficits that negatively impact quality of life. GD3, an oncofoetal ganglioside is re-expressed in medulloblastoma and is thought to be associated with mitochondrially-mediated apoptosis. 9-O-acetyl GD3 (GD3A), its acetylated form, has been shown to protect cells from GD3-mediated apoptosis. Sialate-O-acetylesterase (SIAE) is responsible for cleaving the acetyl group from GD3A. A genome-wide transcriptional analysis of 103 MB patients revealed that this enzyme is significantly down-regulated in MB tissue compared to non-neoplastic foetal and adult brain (p>0.02). To determine if this pathway may serve as a potential therapeutic target we used a well described tet-responsive inducible expression system. SIAE and a mutant of that has been shown to have very little catalytic activity (SIAE-S127A) were expressed in the RES256 cell line. SIAE and SIAE-S127A expression was verified by Western blot analysis after 48 hours of induction with low levels of doxycycline. SIAE expression was also confirmed by SIAE-S127A expressing RES256 cells in conditioned media, but was not secreted by wild-type expressing cells. Over-expression of SIAE showed a significant increase in GD3 expression (p<0.05), and a significant increase in depolarisation of the mitochondrial membrane potential (p<0.05). These changes were not seen on induction of expression of a catalytic mutant SIAE-S127A which has been shown to have almost no catalytic activity, nor by vector controls. With induction of SIAE expression and with etoposide treatment there was a significant reduction in IC50 in SIAE overexpressing clones after 48 hours (p<0.01). After 72 hours of treatment there was a reduction in IC50 (p<0.001). In SIAE-S127A expressing cells there was also a significant reduction in IC50 after 72 hours of treatment (p<0.05). Changes in IC50 for empty vector controls were not significant (p>0.05). Our results suggest that SIAE over-expression leads to a collapse of the mitochondrial membrane potential in RES256 cells. Additionally, these results suggest that SIAE over-expression may sensitize cells to chemotherapeutics, potentially reducing side-effects.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.013

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.250
Teacher spread0.242 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2016
Admission routes1
Has abstractyes

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