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Record W2791746799 · doi:10.1113/ep086822

ATP increases [Ca<sup>2+</sup>]<sub>i</sub>and activates a Ca<sup>2+</sup>‐dependent Cl<sup>−</sup>current in rat ventricular fibroblasts

2018· article· en· W2791746799 on OpenAlexafffund
Noriyuki Hatano, Susumu Ohya, Yuji Imaizumi, Robert B. Clark, Darrell D. Belke, Wayne R. Giles

Bibliographic record

VenueExperimental Physiology · 2018
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicAdenosine and Purinergic Signaling
Canadian institutionsUniversity of Calgary
FundersCanadian Institutes of Health ResearchAlberta Innovates - Health SolutionsHeart and Stroke Foundation of Canada
KeywordsNiflumic acidPurinergic receptorPPADSDIDSP2Y receptorReversal potentialBiophysicsChannel blockerPatch clampIntracellularChemistryUridine triphosphateElectrophysiologyMembrane potentialPhospholipase CAgonistCell biologyInternal medicineBiologyExtracellularBiochemistryCalciumReceptorNucleotideMedicine

Abstract

fetched live from OpenAlex

New Findings What is the central question of this study? Although electrophysiological and biophysical characteristics of heart fibroblasts have been studied in detail, their responses to prominent paracrine agents in the myocardium have not been addressed adequately. Our experiments characterize changes in cellular electrophysiology and intracellular calcium in response to ATP. What is the main finding and its importance? In rat ventricular fibroblasts maintained in cell culture, we find that ATP activates a specific subset of Ca2+‐activated Cl−channels as a consequence of binding to P2Y purinoceptors and then activating phospholipase C. This response is not dependent on [Ca2+]obut requires an increase in [Ca2+]iand is modulated by the type of nucleotide that is the purinergic agonist. Abstract Effects of ATP on enzymatically isolated rat ventricular fibroblasts maintained in short‐term (36–72 h) cell culture were examined. Immunocytochemical staining of these cells revealed that a fibroblast, as opposed to a myofibroblast, phenotype was predominant. ATP, ADP or uridine 5′‐triphosphate (UTP) all produced large increases in [Ca2+]i. Voltage‐clamp studies (amphotericin‐perforated patch) showed that ATP (1–100 μm) activated an outwardly rectifying current, with a reversal potential very close to the Nernst potential for Cl−. In contrast, ADP was much less effective, and UTP produced no detectable current. The non‐selective Cl−channel blockers niflumic acid, DIDS and NPPB (each at 100 μm), blocked the responses to 100 μmATP. An agonist for P2Y purinoceptors, 2‐MTATP, activated a very similar outwardly rectifying C1−current. The P2Y receptor antagonists, suramin and PPADS (100 μmeach), significantly inhibited the Cl−current produced by 100 μmATP. ATP was able to activate this Cl−current when [Ca2+]owas removed, but not when [Ca2+]iwas buffered with BAPTA‐AM. In the presence of the phospholipase C inhibitor U73122, this Cl−current could not be activated. PCR analysis revealed strong signals for a number of P2Y purinoceptors and for the Ca2+‐activated Cl−channel, TMEM16F (also denoted ANO6). In summary, these results demonstrate that activation of P2Y receptors by ATP causes a phospholipase C‐dependent increase in [Ca2+]i, followed by activation of a Ca2+‐dependent Cl−current in rat ventricular fibroblasts.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.253
Teacher spread0.245 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2018
Admission routes2
Has abstractyes

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