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Record W2981074229 · doi:10.1016/j.jalz.2019.08.071

P4‐524: PROTEOLYTIC CLEAVAGE OF TAU IN CORTICOBASAL DEGENERATION AND PROGRESSIVE SUPRANUCLEAR PALSY PATHOGENESIS

2019· article· en· W2981074229 on OpenAlexaff
James P. Quinn, Nicola J. Corbett, Walter Nickel, Katherine A. B. Kellett, Nigel M. Hooper

Bibliographic record

VenueAlzheimer s & Dementia · 2019
Typearticle
Languageen
FieldMedicine
TopicAlzheimer's disease research and treatments
Canadian institutionsDiscovery Centre
Fundersnot available
KeywordsProgressive supranuclear palsyTauopathyGranzymeTau proteinCorticobasal degenerationChemistryBiologyMolecular biologyCell biologyBiochemistryPerforinNeurodegenerationAlzheimer's diseaseGeneticsIn vitroMedicinePathology

Abstract

fetched live from OpenAlex

During the pathogenesis of tauopathies, such as corticobasal degeneration (CBD) and progressive supranuclear palsy (PSP), proteases cleave tau into fragments which have an altered propensity to be phosphorylated, to spread, to aggregate and propagate aggregation. One protease responsible to produce neurotoxic tauopathy-specific tau fragments and its potential role in tauopathy pathogenesis was investigated. The MEROPS database was used to identify the programmed cell death activating protease granzyme A as a candidate to cleave tau in a region known to produce tauopathy-specific tau fragments. The cleavage specificity was assayed in vitro using recombinant proteins, analysed by gel electrophoresis, western blotting with epitope-specific tau antibodies and mass spectrometry. HEK-293 cells expressing granzyme A and tau were employed to identify whether these cleavage events occur intracellularly. Cleavage sites identified by mass spectrometry were inhibited by site-directed mutagenesis. Granzyme A-cleaved tau fragments were expressed in HEK-293 and CHO-K1 cells to analyse transcellular spreading mechanisms, phosphorylation status, aggregation propensity and templated-seeded aggregation. Finally, the tau phosphorylation and fragmentation pattern were analysed in CBD and PSP patient brains. Through a series of experiments, we confirmed that full-length tau1-441 is cleaved by granzyme A at R194-S195 and R209-S210 to produce tauopathy-specific tau fragments. Granzyme A-cleaved C-terminal tau fragments have increased levels of phosphorylation, are more prone to aggregate and propagate aggregation compared to tau1-441, while these phenotypes are decreased in the granzyme A-cleaved N-terminal tau fragments. However, the latter demonstrate an increased propensity to undergo transcellular spreading compared to granzyme A-cleaved C-terminal tau fragments and full-length tau1-441. Furthermore, increased amounts of cleaved, phosphorylated and aggregated tau were identified in CBD and PSP patient brains compared to age-matched controls. Cleavage sites within tau to produce granzyme A-cleaved tau fragments were identified by mass spectrometry in CBD and PSP patient brains. Our results show that (i) granzyme A cleaves tau to produce tauopathy-specific tau fragments, and (ii) granzyme A-cleaved tau fragments have distinct cellular effects which could play an important role in tauopathy pathogenesis. Elucidating granzyme A-cleavage of tau is crucial in unravelling tauopathy pathogenesis to identify potential therapeutic targets and diagnostic biomarkers.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0020.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.021
GPT teacher head0.288
Teacher spread0.267 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes1
Has abstractyes

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