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Record W3154980607 · doi:10.1007/s00401-021-02296-1

TREM2 expression in the brain and biological fluids in prion diseases

2021· article· en· W3154980607 on OpenAlexafffund
Daniela Díaz‐Lucena, Niels Kruse, Katrin Thüne, Matthias Schmitz, Anna Villar‐Piqué, José Eriton Gomes da Cunha, Péter Hermann, Óscar López-Pérez, Pol Andrés‐Benito, Anna Ladogana, Miguel Calero, Enríc Vidal, J. Riggert, Hailey Pineau, Valerie L. Sim, Henrik Zetterberg, Kaj Blennow, José Antonio del Rı́o, Alba Marín‐Moreno, Juan Carlos Espinosa, Juan María Torres, Raquel Sánchez‐Valle, Brit Mollenhauer, Isidró Ferrer, Inga Zerr, Franc Llorens

Bibliographic record

VenueActa Neuropathologica · 2021
Typearticle
Languageen
FieldNeuroscience
TopicNeuroinflammation and Neurodegeneration Mechanisms
Canadian institutionsUniversity of Alberta
FundersInterregInstituto de Salud Carlos IIIHjärnfondenUK Dementia Research InstituteVetenskapsrådetMinistero della SaluteEuropean Regional Development FundEuropean CommissionUniversity of AlbertaCentro de Investigación Biomédica en Red sobre Enfermedades NeurodegenerativasAlzheimer's Drug Discovery FoundationCentres de Recerca de CatalunyaPolarforskningssekretariatetMinisterio de Ciencia, Innovación y UniversidadesAlzheimer SocietyEU Joint Programme – Neurodegenerative Disease ResearchMinisterio de Ciencia e InnovaciónGeneralitat de CatalunyaFundació la Marató de TV3
KeywordsTREM2PRNPEctodomainADAM10ReceptorBiologyMicrogliaAlzheimer's diseaseImmunologyCerebrospinal fluidMedicinePathologyDiseaseInflammationInternal medicineDisintegrinPrion proteinMetalloproteinase

Abstract

fetched live from OpenAlex

Triggering receptor expressed on myeloid cells 2 (TREM2) is an innate immune cell surface receptor that regulates microglial function and is involved in the pathophysiology of several neurodegenerative diseases. Its soluble form (sTREM2) results from shedding of the TREM2 ectodomain. The role of TREM2 in prion diseases, a group of rapidly progressive dementias remains to be elucidated. In the present study, we analysed the expression of TREM2 and its main sheddase ADAM10 in the brain of sporadic Creutzfeldt-Jakob disease (sCJD) patients and evaluated the role of CSF and plasma sTREM2 as a potential diagnostic marker of prion disease. Our data indicate that, compared to controls, TREM2 is increased in sCJD patient brains at the mRNA and protein levels in a regional and subtype dependent fashion, and expressed in a subpopulation of microglia. In contrast, ADAM10 is increased at the protein, but not the mRNA level, with a restricted neuronal expression. Elevated CSF sTREM2 is found in sCJD, genetic CJD with mutations E200K and V210I in the prion protein gene (PRNP), and iatrogenic CJD, as compared to healthy controls (HC) (AUC = 0.78-0.90) and neurological controls (AUC = 0.73-0.85), while CSF sTREM2 is unchanged in fatal familial insomnia. sTREM2 in the CSF of cases with Alzheimer's disease, and multiple sclerosis was not significantly altered in our series. CSF sTREM2 concentrations in sCJD are PRNP codon 129 and subtype-related, correlate with CSF 14-3-3 positivity, total-tau and YKL-40, and increase with disease progression. In plasma, sTREM2 is increased in sCJD compared with HC (AUC = 0.80), displaying positive correlations with plasma total-tau, neurofilament light, and YKL-40. We conclude that comparative study of TREM2 in brain and biological fluids of prion diseases reveals TREM2 to be altered in human prion diseases with a potential value in target engagement, patient stratification, and disease monitoring.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.003
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.172
Threshold uncertainty score0.400

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.003
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.050
GPT teacher head0.274
Teacher spread0.223 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations31
Published2021
Admission routes2
Has abstractyes

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