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Record W4308108258 · doi:10.1016/j.ccell.2022.10.002

Selective advantage of epigenetically disrupted cancer cells via phenotypic inertia

2022· article· en· W4308108258 on OpenAlexfundno aff
Ioannis Loukas, Fabrizio Simeoni, Marta Milan, Paolo Inglese, Harshil Patel, Robert Goldstone, Philip East, Stephanie Strohbuecker, Richard Mitter, Bhavik Talsania, Wenhao Tang, Colin D.H. Ratcliffe, Erik Sahai, Vahid Shahrezaei, Paola Scaffidi

Bibliographic record

VenueCancer Cell · 2022
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer Genomics and Diagnostics
Canadian institutionsnot available
FundersMedical Research CouncilFonds de Recherche du Québec - SantéMerck Sharp and DohmeFonds de recherche du QuébecWellcome TrustFrancis Crick InstituteCancer Research UK
KeywordsPhenotypeBiologyCancer cellCancerCell biologyGeneticsComputational biologyGene

Abstract

fetched live from OpenAlex

The evolution of established cancers is driven by selection of cells with enhanced fitness.Subclonal mutations in numerous epigenetic regulator genes are common across cancer types, yet their functional impact has been unclear.Here, we show that disruption of the epigenetic regulatory network increases the tolerance of cancer cells to unfavorable environments experienced within growing tumors by promoting the emergence of stress-resistant subpopulations.Disruption of epigenetic control does not promote selection of genetically defined subclones or favor a phenotypic switch in response to environmental changes.Instead, it prevents cells from mounting an efficient stress response via modulation of global transcriptional activity.This ''transcriptional numbness'' lowers the probability of cell death at early stages, increasing the chance of long-term adaptation at the population level.Our findings provide a mechanistic explanation for the widespread selection of subclonal epigenetic-related mutations in cancer and uncover phenotypic inertia as a cellular trait that drives subclone expansion.Disruption of the epigenetic regulatory network enhances cell fitness under environmental stress In physiology, epigenetic mechanisms mediate the cellular response to environmental cues.We therefore asked whether disruption of epigenetic control in cancer cells may affect their interactions with the tumor microenvironment (TME).We selected two cancer types originating from distinct lineages: NSCLC lung (D) Visualization of subclonal mutations affecting epigenetic regulator genes in the TRACERx patient cohort.Genes grouped by functional class.Colors legend as in (A).(E) Ratio of nonsynonymous over synonymous mutations in individual genes in the indicated groups.N = 190, 65, and 10,025 genes, respectively.Red lines denote median values.p values relative to the rest of the genes from one-tailed Student's t test after random sampling.(F) Phylogenetic trees visualizing emerging subclones in two tumors.Mutated epigenetic regulator genes identified in the trunk (gray line, clonal mutations) or in the branches (black lines, subclonal mutations) are indicated.Genes color-coded as in (A).See also Figure S1 and Table S1. DECLARATION OF INTERESTSH.P. is currently an employee of Seqera Labs.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.001
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.005
GPT teacher head0.238
Teacher spread0.233 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations46
Published2022
Admission routes1
Has abstractyes

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