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Record W4406222527 · doi:10.1002/alz.094584

Driving Tau Pathogenicity Seeding ‐ How Fibril Structure and Post‐Translational Modifications Alter Seeding Capacity

2024· article· en· W4406222527 on OpenAlexaff
Alysa Kasen, Libby Breton, Lindsay Meyerdirk, Sofia Lövestam, Jacob A. McPhail, Ariel Louwrier, Sjors H. W. Scheres, Michael X. Henderson

Bibliographic record

VenueAlzheimer s & Dementia · 2024
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicPrion Diseases and Protein Misfolding
Canadian institutionsAptose Biosciences (Canada)
Fundersnot available
KeywordsSeedingFibrilBiophysicsChemistryBiologyAgronomy

Abstract

fetched live from OpenAlex

Abstract Background The accumulation of hyperphosphorylated, aggregated tau in neurons is one of the hallmarks of Alzheimer’s disease (AD). Recent work in structural biology has solved the structure of tau fibrils in several tauopathies and found that the structure of the tau fibrils varies between diseases, but fibril structure is conserved among patients within the same disease, suggesting fibril structure relates to its pathogenicity. Tau fibrils derived from AD brain (AD PHFs) seed AD‐like pathology in wild‐type mice, yet efforts to recapitulate this seeding with recombinant fibrils have failed. The differential capacity of tau fibrils to seed pathology also supports the relevance of tau structure to its pathogenicity. We hypothesized that recombinant fibrils that recapitulate the core region structure of AD tau will show similar seeding capacity to AD tau, with PTMs playing a modulatory role. Methods We took advantage of recently developed recombinant tau fibrils with diverse structures to investigate how tau fibril structure and PTMS are related to tau seeding capacity in primary cortical neurons. Fibrils of interest from the screen were then assessed through hippocampal injection into wild‐type and MAPT knock‐in mice. Results In‐vitro screening showed that fibrils more closely resembling the core structure of an AD PHF had a higher seeding capacity than other fibrils. However, the replication of the core structure alone in truncated fibrils is not sufficient to fully replicate the seeding capacity of AD PHFs, indicating that the region outside of the core structure, containing PTMs, likely plays an essential role in pathology seeding. This finding was replicated in the seeding model into wild‐type and MAPT KI mice. We also found that tau fibrils containing PTMs had a higher seeding capacity than full length fibrils without PTMs. However, the presence of PTMs alone was not enough to fully replicate the seeding capacity of AD PHFs. Conclusions The structure and PTM patterns of tau fibrils appear to be closely tied to fibril pathogenicity, with recombinant fibrils more closely resembling AD PHFs having a more similar seeding capacity. We believe that the generation of fibrils closely resembling AD PHFs can lead to improved model systems of tau pathology in AD.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.249
Teacher spread0.231 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2024
Admission routes1
Has abstractyes

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