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Record W4409631974 · doi:10.1158/1538-7445.am2025-2171

Abstract 2171: Amezalpat, a peroxisome proliferator-activated receptor alpha (PPARα) antagonist, inhibits suppressive macrophage development, activation and function

2025· article· en· W4409631974 on OpenAlexaff
Mandy I. Cheng, Ademola Esomojumi, Dara Burdette, Valerie Chen, Sam H. Whiting, Nathan Standifer

Bibliographic record

VenueCancer Research · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicPeroxisome Proliferator-Activated Receptors
Canadian institutionsMcGill Genome Centre
Fundersnot available
KeywordsPeroxisome proliferator-activated receptorPeroxisomeAntagonistChemistryReceptorAlpha (finance)Function (biology)Peroxisome proliferatorPeroxisome proliferator-activated receptor alphaCell biologyPharmacologyEndocrinologyCancer researchBiologyMedicineBiochemistryNuclear receptorTranscription factorGene

Abstract

fetched live from OpenAlex

Amezalpat (TPST-1120) is a first-in-class, small molecule competitive antagonist of peroxisome proliferator-activated receptor alpha (PPARα), the master transcriptional regulator of fatty acid oxidation (FAO). Analysis of biomarker data from a previous Phase I study demonstrated amezalpat dose-dependent changes in immune genes associated with myeloid populations. Suppressive M2 macrophages are a prominent myeloid population associated with poor prognosis in many cancers. As M2 macrophages are reliant on FAO, we sought to assess the effects of amezalpat on development, activation and function of these cells. Flow cytometry analysis of M2 macrophages polarized in the presence of amezalpat displayed concentration-dependent reductions in canonical M2 macrophage markers CD163, CD206, and Arg-1 (p<0.05 by Wilcoxon’s test). ELISA measurement of cytokine production by amezalpat-treated M2 macrophages demonstrated a statistically significant reduction in the suppressive cytokines IL-10 (n=8; p<0.05 by Wilcoxon’s test) and TGF-β (n=3 by One-way ANOVA), suggesting amezalpat-induced functional impairment of this population. Additionally, amezalpat treatment of M2 macrophages caused significant reductions in mitochondrial mass, measured with a mitochondria-specific dye by flow cytometry (p<0.01 by two-sample T test). Finally, in co-cultures of M2 macrophages with autologous activated CD8+ T cells and HCC tumor cells (SNU-449), amezalpat induced tumor cell cytotoxicity and M2 cell death commensurate with cell-type specific expression levels of its target PPARα. Together, these data suggest amezalpat modulates suppressive macrophage development and function and supports the contribution of an immune-mediated mechanism to anti-tumor activity reported in clinical studies. Citation Format: Mandy I. Cheng, Ademola Esomojumi, Dara Burdette, Valerie Chen, Jorg H. Fritz, Sam Whiting, Nathan Standifer. Amezalpat, a peroxisome proliferator-activated receptor alpha (PPARα) antagonist, inhibits suppressive macrophage development, activation and function [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 1 (Regular Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_1):Abstract nr 2171.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score0.019

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0060.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.027
GPT teacher head0.340
Teacher spread0.313 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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