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Record W4411846582 · doi:10.3899/jrheum.2025-0314.1

Macrophage Migration Inhibitory Factor and Hypoxia-Inducible Factor 1-Alpha May Play a Role in Maintaining Intestinal Homeostasis Through Regulation of Occludin and Claudin-1 Expression

2025· article· en· W4411846582 on OpenAlexaffvenue
Shaghayegh Foroozan-Boroojeni, Akihiro Nakamura, Mansi K. Aparnathi, Mariia Korshko, Nigil Haroon

Bibliographic record

VenueThe Journal of Rheumatology · 2025
Typearticle
Languageen
FieldImmunology and Microbiology
TopicMacrophage Migration Inhibitory Factor
Canadian institutionsQueen's UniversityUniversity Health Network
Fundersnot available
KeywordsMacrophage migration inhibitory factorOccludinMedicineHomeostasisClaudinImmunologyHypoxia (environmental)Inhibitory postsynaptic potentialHypoxia-inducible factorsAlpha (finance)Cell biologyEndocrinologyCytokineBiologyTight junctionBiochemistryGeneSurgeryChemistry

Abstract

fetched live from OpenAlex

Objectives Axial spondyloarthritis (axSpA) is a chronic inflammatory condition mainly impacting the musculoskeletal system. The majority of axSpA patients experience gut inflammation, with 60% having microscopic changes and 10% having overt inflammatory bowel disease (IBD). Over the years, researchers have linked many pathways and cell populations to the pathogenesis of gut inflammation in axSpA. One notable player may be the macrophage migration inhibitory factor (MIF), a crucial factor in axSpA development. In a SpA mouse model (SKG mice), MIF overexpression mirrors key clinical features of axSpA, while inhibiting or depleting MIF attenuates these symptoms. It is believed that MIF and hypoxia-inducible factor 1 alpha (HIF-1α) have a functional interdependence. Nevertheless, the precise role of MIF and HIF in gut inflammation in axSpA remains uncertain. Here we aimed to investigate the MIF-HIF interaction’s impact on gut homeostasis and inflammation in the SKG mouse model. Methods Eight weeks old SKG mice were used for this 4-arm study, with 3 mice per arm: SKG control mice, SKG mice treated with curdlan, SKG-MIF Knock Out (KO), and SKG-MIFKO mice treated with curdlan. After 8 weeks of curdlan/vehicle treatment, 16 weeks old mice were euthanized, and formalin-fixed paraffin-embedded (FFPE) blocks of ileum were prepared for immunohistochemistry (IHC). IHC was performed to assess the tight junction markers, occludin and claudin-1 and, HIF-1α expression. Kruskal-Wallis test was used to analyze the difference between the 4 groups. Results SKG-MIFKO mice showed reduced occludin and claudin-1 expression, indicating tight junction disruption (Figure 1). HIF-1α expression decreased significantly in SKG-MIFKO and SKG-MIFKO+curdlan groups. Notably, HIF-1α expression decreased further in SKG-MIFKO+curdlan compared to SKG+curdlan, with a trend of higher expression in SKG+curdlan compared to controls (Figure 1). Conclusion Knocking out MIF in SKG mice has resulted in a notable improvement in axSpA symptoms. However, our findings show that MIF has a protective role in the gut of SKG mice, which is unlike what we see in joints. In the absence of MIF, there was a decrease in the expression of tight junction markers and HIF-1 α, which suggests that the interaction between MIF and HIF is essential for maintaining the integrity of the gut epithelial barrier in the SKG mouse model of SpA. Further investigation is required to determine the role of HIF-1α in transcriptional regulation of tight junction markers.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.259
Teacher spread0.244 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2025
Admission routes2
Has abstractyes

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