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Record W4412951835 · doi:10.1002/1878-0261.70107

Investigating the cell of origin and novel molecular targets in Merkel cell carcinoma: a historic misnomer

2025· article· en· W4412951835 on OpenAlexafffund
Richie Jeremian, Sriraam Sivachandran, Melissa A. Galati, Brandon Ramchatesingh, Hibo Rijal, Johnny Hanna, Elena Netchiporouk, May Chergui, Margaret Redpath, Samy Abou Setah, Ivan V. Litvinov

Bibliographic record

VenueMolecular Oncology · 2025
Typearticle
Languageen
FieldMedicine
TopicPolyomavirus and related diseases
Canadian institutionsSt Mary's Hospital CentreUniversité LavalMcGill UniversityQueen's UniversitySt. Mary's UniversityUniversity of TorontoMcGill University Health Centre
FundersInstitute of Cancer ResearchFonds de Recherche du Québec - SantéCancer Research Society
KeywordsMerkel cell polyomavirusMerkel cell carcinomaBiologyTranscriptomeMerkel cellCell typeCellDownregulation and upregulationImmune systemCancer researchGeneImmunologyGene expressionGeneticsCarcinoma

Abstract

fetched live from OpenAlex

Merkel cell carcinoma (MCC) is a highly aggressive disease with the poorest prognosis among skin cancers, originally posited to be derived from Merkel cells. Emerging evidence, however, suggests other potential origins for MCC, including hematological lineages. We utilized targeted and multi-omics approaches to explore gene expression patterns at protein and RNA levels of MCCs. Western blotting, immunofluorescence, and immunohistochemistry were performed using fresh and 92 FFPE samples of primary and metastatic MCC, and two MCC cell lines (MS-1, HaCaT). RNA sequencing of selected FFPE samples identified differentially expressed genes based on sex and Merkel cell polyomavirus (MCPyV) status. Finally, weighted gene correlation network analysis (WGCNA) and cell type enrichment analyses were employed to determine pathway and cell type enrichment, respectively. MCC patient samples heterogeneously expressed B-cell and neuroendocrine markers and novel molecular targets including BCMA, CD10, CD93, PAX5, TdT, IgA, and CD19. Transcriptome analysis demonstrated differentially expressed genes based on sex and MCPyV status. MCPyV+ tumors had significant upregulation of genes involved in immune cell function and downregulation of processes related to neuronal activity. WGCNA highlighted enrichment for pathways involved in immune function, including B-cell differentiation. Cell type enrichment analysis highlighted enrichment for multipotent stem cells, several immune cell types, and keratinocytes. Our findings support previous studies which confirm that MCC is unlikely to be derived from Merkel cells and instead from multiple or divergent cell types, including those of B-cell lineage. Our work highlights the need for a more personalized approach to diagnosis/characterization and treatment of MCCs, given the documented variability of novel potentially targetable pathways.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.141
Threshold uncertainty score0.486

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.011
GPT teacher head0.273
Teacher spread0.262 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2025
Admission routes2
Has abstractyes

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