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Record W7139714965

Studies of Microsatellite Instability Signatures in Replication Repair Deficient Malignant and Germline Tissues

2025· dissertation· W7139714965 on OpenAlexaboutno aff
Logine Negm

Bibliographic record

VenueTSpace (University of Toronto) · 2025
Typedissertation
Language
FieldMedicine
TopicGenetic factors in colorectal cancer
Canadian institutionsnot available
Fundersnot available
KeywordsMicrosatellite instabilityDNA mismatch repairGermlineGenome instabilityGermline mutationMicrosatelliteCancer
DOInot available

Abstract

fetched live from OpenAlex

DNA replication errors are resolved by the mismatch repair (MMR) proteins (MLH1, MSH2, MSH6, or PMS2) although pathogenic variants in MMR genes result in mismatch repair deficiency (MMRD). MMRD arises through primary causes (somatic or germline variants), or secondary causes (treatment-induced). Monoallelic germline variants in MMR genes causes Lynch Syndrome in adults who develop primarily gastrointestinal cancers. Biallelic germline MMR variants cause constitutional mismatch repair deficiency (CMMRD) in children who develop predominantly central nervous system malignancies. MMRD induces extreme genomic instability causing cancers characterized by hypermutation, microsatellite instability (MSI), and resistance to chemoradiation. The unique biology of MMRD tumors and high likelihood of cancer development means accurate diagnosis is crucial. MMRD diagnosis has relied on several methods which are insufficient in non-colorectal and childhood cancers. Fortunately, signatures of MSI provide highly specific diagnosis.Leveraging millions of microsatellites across the genome, this thesis resulted in creation of the functional assay LOw-pass Genomic Instability Characterization (LOGIC) for robust MMRD diagnosis in tumors. LOGIC additionally achieved diagnosis of CMMRD using germline tissues and MSI analysis revealed biological insights into MMRD mutagenesis. MSI correlated with specific MMR variants, tissues, and age at initial cancer diagnosis, enabling tailored screening protocols for the future. The capacity to reliably diagnose MMRD enabled determination of its prevalence in primary gliomas, which represent the leading cause of cancer-related mortality in children. A population-based screen of gliomas in children, adolescents, and young adults (CAYA, 0-40 years) from Toronto, St Jude Children’s Research Hospital, and the Children’s Brain Tumor Network revealed a significant prevalence in high-grade gliomas. MMRD was enriched in IDH- and H3-WT high-grade gliomas but was absent in gliomas with pediatric-type alterations, fusions, or copy number variants. Almost all MMRD gliomas were driven by primary causes, namely germline alterations in MMR genes causing Lynch Syndrome, redefining the cancer spectrum in Lynch Syndrome. Immunotherapy in a cohort from the International Replication Repair Deficiency Consortium revealed significantly improved overall survival in MMRD CAYA gliomas. We suggest increased MMRD screening in specific glioma subtypes, germline testing for patients and family members, and further studies of immunotherapies in CAYA.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.313
Teacher spread0.288 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

Explore more

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